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臀肌腱撕裂与修复

Updated Sep 2026
Illustration: hip

本页面由机器翻译,尚未经临床医生审核。英文版本为权威版本。

为何建议进行此手术

臀肌肌腱撕裂(这些肌腱连接着在您行走时保持骨盆稳定的肌肉)表现为髋部外侧的钝痛,且在受压、站立及外展腿部时疼痛加剧。

这些撕裂通常源于随时间推移的逐渐磨损,但跌倒也可能导致撕裂。我们通常首先采用非手术治疗,如改变活动方式、物理治疗或注射治疗,当这些方法未能带来足够改善时,我们才会考虑手术。当影像学扫描和体格检查均显示肌腱撕裂且髋部肌力减弱时,我们会提供修复手术。对于症状严重的患者,在回顾性病例中,修复手术使 95% 的患者症状得到缓解。目标是减轻疼痛、增强肌力,并使您在行走时髋部更加稳定。我们将共同讨论这一决定。

术前

一旦手术计划确定,我们将为您提供明确的指示,以确保手术当天顺利进行。您需要提前七小时停止进食和饮水。我们要求七小时而非更短的时间,以便如果手术室排程提前,您的手术可以提前进行。某些药物可能需要暂停,我们会告知您具体哪些药物以及何时暂停。请携带一份您正在服用的所有药物的书面清单,安排有人开车送您回家,并穿着宽松舒适的衣物。已安排的检查,如X光、MRI或超声,有助于我们规划手术。如果您有其他健康状况,可能需要进行血液检查或与麻醉师进行会诊。

手术当日

您将抵达医院的手术入院单元,在此办理入院手续并进行术前准备。您将见到麻醉医生,即负责让您入睡并在手术期间保持您舒适状态的医生。本手术在全身麻醉下进行。有时会追加区域神经阻滞以缓解术后疼痛;麻醉医生将在手术当日就此与您沟通。

随后,您将被带入手术室进行手术。手术结束后,您将在复苏区苏醒。在麻醉药效消退期间,护士将全程监护您。待您的生命体征稳定后,根据手术类型及恢复情况,您将被转入病房或直接回家。

手术内容

手术的目的是将撕裂的臀肌腱重新附着到髋部外侧的骨骼上。您的外科医生将在两种手术方式中选择一种,这两种方式均通过髋部外侧的同一部位或围绕该部位进行操作。

一种选择是微创手术,也称为内窥镜修复。这种方法使用几个小切口,而不是一个大切口。一根细长的摄像头通过其中一个切口插入,以便外科医生在屏幕上看到撕裂的肌腱。通过其他小切口,外科医生清除受损组织,并将肌腱缝合固定在骨骼上。由于切口较小,这种方法对周围肌肉的干扰较少。对于部分撕裂,可以通过肌腱本身进行缝合修复,从而保持肌腱健康的外层部分与骨骼的连接不受影响。

另一种选择是开放修复。这种方法在髋部外侧使用一个较大的切口,以便外科医生直接看到并触及肌腱。撕裂的肌腱使用牢固的不可吸收缝线缝合回骨骼上。如果撕裂较大,或者肌腱回缩过远而无法触及骨骼,外科医生可能会添加一块组织补丁或转移附近的肌腱来桥接间隙并加固修复。

如果发现有其他问题,如撕裂的盂唇(髋臼周围的软骨边缘),这两种方法都可以与髋关节镜手术(关节内部的微创观察)结合使用。切口用缝线关闭,并覆盖敷料。

术后

您将在复苏室醒来,随着麻醉药效消退,护士会密切观察您的情况。您的髋部会感到疼痛,我们会为您提供镇痛治疗以确保您的舒适。髋部切口一侧的缝合处将覆盖敷料。敷料通常保留约10天;除非我们告知您,否则请勿在此之前自行拆除。我们会在复诊时为您更换或拆除敷料。大多数人当天即可站立,并在助行器或拐杖的辅助下行走几步,物理治疗师会向您演示方法。最初24小时应有人陪同。您的医疗团队会告知您是当天出院还是在医院过夜。

恢复

在最初几天,您的髋部会感到疼痛,缝合处周围可能会感觉肿胀。止痛药有助于缓解不适,随着时间推移,疼痛通常会逐渐减轻。在膝盖下方垫一个枕头休息可能会有所帮助。用毛巾包裹的冰袋也有助于减轻肿胀。

大多数人在手术当天即可借助拐杖或助行器站立并行走几步。您的物理治疗师会指导您了解腿部可以承受的重量,并引导您进行温和的练习。这些练习有助于在肌腱愈合期间保持髋部活动和肌肉功能。您将在家中进行这些练习,循序渐进,并根据髋部的恢复情况逐步增加强度。

在家中,您可以小心地活动并进行日常事务,但在医生告知安全之前,请避免对疼痛侧施加过大压力、深度弯曲或提起重物。早期仰卧或向非手术侧侧卧通常更为舒适。一旦肿胀消退且行走感觉稳定,您将从使用拐杖过渡到独立行走。当您的外科医生对您的力量和活动能力感到满意时,您可以恢复驾驶和日常活动。

每个人的恢复情况各不相同。您的时间表可能有所不同,您的外科医生和物理治疗师将在每次复诊时为您提供指导。

可能出现的问题

大多数患者恢复良好,但偶尔会出现问题。您的外科医生和团队会密切监测您,以便尽早发现任何问题。

我们重点关注的常见问题是肌腱在缝合修复后再次撕裂。这被称为再撕裂。您可能会注意到髋部外侧疼痛复发,有时伴有术前曾感受到的同样钝痛,或者在行走时感觉髋部无力或失稳。如果疼痛在缓解后再次出现,请告知我们。我们将检查您的髋部并安排影像学检查,以确认肌腱是否保持完好。如果撕裂复发且疼痛持续,有时可能需要再次手术进行修复。请在下次复诊时告知我们任何复发的疼痛,如果疼痛让您困扰,请尽早致电诊所。

偶尔,修复可能需要第二次手术来固定。这被称为翻修手术。当肌腱再次撕裂且疼痛复发时,会发生这种情况。体征相同:原本缓解的髋部外侧疼痛、站立或行走时的无力感,以及脚下稳定性降低。如果您注意到这些情况,请立即联系我们,不要等待。我们将评估您的情况并讨论变化。

一些接受髋关节置换术而非修复术的患者也可能出现臀肌肌腱问题。如果您正在接受髋关节置换,且臀肌肌腱磨损或撕裂,术后您可能会注意到髋部外侧疼痛增加,且在恢复期间髋部可能感觉不如预期稳定或舒适。如果影像学检查显示此情况,我们将在术前与您讨论,并在术后复诊时密切关注您髋部外侧的情况。

本页上的并发症表列出了典型发生率,如果您想了解具体细节,请参阅该表。

何时联系我们

大多数问题会在早期显现,我们更希望尽早获知。如果您的疼痛在加剧而非缓解,伤口周围皮肤发红、发热或渗出液体,或您感到发热,请致电我们。如果您出现突发剧烈疼痛、小腿肿胀、呼吸急促、腿部麻木或无法活动腿部,请立即前往急诊。这些症状可能提示血栓或神经问题,需立即检查。


Evidence & references

This is the clinical evidence summary written for health professionals. It is technical, and it lists the research this page was built from. You do not need to read it to understand your treatment or to make a decision about it.

Anatomy & Pathophysiology

Bony Anatomy

  • The hip is a multiaxial joint formed by the articulation between the pelvis and femur, connecting the axial skeleton and the lower extremity [8].
  • The hemipelvis comprises three bones: the ilium, ischium, and pubis, which unite at the triradiate cartilage within the concave acetabulum [8].
  • The acetabulum comprises an articular crescent-moon–shaped lunate surface and a nonarticular central fossa that serves as the attachment point for the ligamentum teres [8].
  • The acetabulum is incomplete inferiorly, forming a notch through which vital blood vessels and nerves pass to supply the joint [8].
  • The femoral head forms two-thirds of a sphere, with a small depression at its center from which the ligamentum teres extends to connect to the acetabular notch [8].
  • The neck-shaft angle of the femur averages 125° [8].
  • Normal version, defined as the head-neck angle in the frontal plane, averages 15 to 20° [8].
  • The acetabulum is normally anteverted 15 degrees and obliquely oriented in the coronal plane 45 degrees caudally [14].
  • The posterosuperior articular surface of the acetabulum is thickened to accommodate weight bearing [14].
  • The inferior surface of the acetabulum contains the acetabular (cotyloid) notch, which is bound by the transverse acetabular ligament [14].
  • The femoral neck is normally anteverted approximately 14 degrees in relation to the femoral condyles, with a range of 1–40 degrees [14].
  • The femoral neck-shaft angle averages 127 degrees, beginning at 141 degrees in the fetus [14].
  • The ilium is a large flat bone forming the majority of the coxal bone, with the iliac crest terminating anteriorly at the anterior superior iliac spine (ASIS) and posteriorly at the posterior superior iliac spine (PSIS) [8].
  • The ischium is a small L-shaped bone forming the posteroinferior margin of the pelvis, with the ischial tuberosity serving as a large attachment site for multiple muscle groups [8].
  • The pubis bone consists of a body and two rami that connect superiorly to the ilium and inferiorly to the ischium to form the obturator foramen [8].
  • The obturator foramen is covered by a strong membrane that provides surface area for muscle attachments [8].
  • The hemipelvises unite anteriorly at the pubic symphyses and articulate posteriorly with the sacral ala to form the sacroiliac (SI) joint [8].
  • The iliac crest is a palpable rim of the ilium and an important site for bone graft harvest, specifically the iliac tubercle 5 cm posterior to the ASIS [14].
  • The ASIS is palpable at the lateral edge of the inguinal ligament and serves as the origin of the sartorius muscle and transverse and internal abdominal muscles [14].
  • The AIIS is less prominent and serves as the origin of the direct head of the rectus femoris and the iliofemoral ligament [14].
  • The PSIS is located 4–5 cm lateral to the S2 spinous process and is an important landmark for posterior iliac crest bone graft harvest [14].
  • The ischial tuberosity serves as the origin of the hamstrings [14].
  • The iliopsoas muscle/tendon traverses a groove between the iliopectineal eminence and the AIIS [14].

Ligaments and Capsule

  • The hip is surrounded by a dense fibrous capsule extending from the periphery of the acetabulum to the intertrochanteric line of the femoral neck [8].
  • The capsule enhances joint stability by preventing translation of the femoral head in the acetabulum [8].
  • The hip capsule attaches anteriorly and posteriorly along the periphery of the acetabulum outside the labrum [9].
  • Inferiorly, the hip capsule is attached to the acetabular labrum [9].
  • The capsule is attached to the femur anteriorly along the intertrochanteric crest [9].
  • On the posterior side, the capsule attaches only partially, such that the basicervical region of the femoral neck and the intertrochanteric region of the femur are not intracapsular [9].
  • The iliofemoral ligament is also known as the Y ligament of Bigelow; it originates at the AIIS and inserts at the intertrochanteric line [9].
  • The iliofemoral ligament is the thickest and strongest of the three main ligaments supporting the hip [8].
  • The medial portion of the iliofemoral ligament connects the anterior inferior iliac spine to the anterior intertrochanteric line, while the lateral portion originates slightly superior to the medial arm and attaches to the anterior greater trochanter [8].
  • The iliofemoral ligament functions to limit external rotation, while in isolation, the lateral arm limits extension of the joint [8].
  • The iliofemoral ligament becomes taut in full extension, preventing anterior dislocation and hyperextension of the hip [9].
  • The ischiofemoral ligament extends from the ischial margin of the acetabulum to the greater trochanter of the femur [8].
  • The ischiofemoral ligament provides support posteriorly and restricts internal rotation motion [8].
  • The pubofemoral ligament extends from the obturator crest of the pubic bone to the femoral neck and acts to limit abduction of the joint [8].
  • Deep fibers from the iliofemoral, ischiofemoral, and pubofemoral ligaments merge to form the zona orbicularis, which circumvents the femoral neck [8].
  • The twisted orientation of the hip ligaments provides a screw mechanism for the hip in full extension [9].
  • The ligamentum teres originates in the cotyloid fossa and attaches on the fovea of the femoral head [9].
  • The ligamentum teres transmits an arterial branch of the posterior division of the obturator artery to the femoral head, which is less significant in adults [5].
  • The sacrospinous ligament creates the upper border of the lesser sciatic foramen and the lower border of the greater sciatic foramen [9].
  • The sacrotuberous ligament creates the inferior border of the lesser sciatic foramen [9].
  • The piriformis muscle and the sciatic nerve exit from the greater sciatic foramen [9].
  • The short external rotator muscles exit from the lesser sciatic foramen [9].

Labrum

  • The acetabular labrum is a fibrocartilaginous ring attached to the rim of the acetabulum that extends the articulating surface area and increases femoral head coverage [8].
  • The labrum is triangular in cross section, which contributes to its ability to create a pressurized seal of the central compartment of the hip during loading [8].
  • Only the external one-third of the labrum contains blood vessels, leaving the majority of the structure avascular and limiting its healing ability following injury [8].
  • The labrum is highly innervated, with the presence of both mechanoreceptors and nociceptors [8].
  • The labrum is absent in the area of the inferior acetabular notch, where the transverse acetabular ligament serves as the continuation of the labrum, connecting the anterior and posterior lunate surfaces of the acetabulum [8].
  • Labral functions include load transmission, maintenance of vacuum seal, regulation of synovial fluid hydrodynamics, and joint lubrication [5].

Muscular Anatomy

  • The abductors of the hip are predominantly the gluteus medius and minimus muscles [16].
  • The gluteus medius has three different components: anterior, middle, and posterior [16].
  • The gluteus medius and minimus muscles function together to maintain and abduct the femur during the stance phase of gait [16].
  • A Trendelenburg lurch is an attempt by the body to compensate for abductor weakness by bringing the center of gravity closer to the hip center, forcing the patient to lean toward the affected side [16].
  • The gluteus maximus originates from the sacrum, the coccyx, and the sacrotuberous ligaments and inserts into the lateral intramuscular septum [16].
  • The hamstring muscles originate on the ischial tuberosity [16].
  • The primary hip flexor muscles are the iliopsoas, rectus femoris, and sartorius muscles [16].
  • The iliopsoas muscle has a large origin along the iliac crest, the iliac fossa, the sacra ala, the iliolumbar ligaments, and the sacroiliac ligaments [16].
  • The rectus femoris crosses the hip joint and the knee joint, with the straight head originating from the AIIS and the reflected head originating from the supra-acetabular tubercle [16].
  • The sartorius muscle originates on the ASIS, crosses the hip and knee joints, and inserts on the medial aspect of the tibia and the pes anserine complex [16].
  • The tensor fasciae latae muscle originates laterally on the anterolateral edge of the iliac crest and its fibers combine with the fasciae latae to form the iliotibial band [16].
  • The action of the tensor fasciae latae is to flex, abduct, and rotate the hip [16].
  • The external rotators of the hip include the obturator internus and externus, superior and inferior gemelli, quadratus femoris, and piriformis muscles [16].
  • The piriformis forms the reference structure for the posterior part of the hip, with structures identified by whether they originate above or below it [16].
  • The superior gluteal nerve and artery exit the pelvis above the piriformis muscle [16].
  • The pudendal nerve, internal pudendal artery, nerve to the obturator internus, posterior femoral cutaneous nerve, sciatic nerve, inferior gluteal nerve, inferior gluteal artery, and nerve to the quadratus femoris all exit the pelvis below the piriformis [16].
  • In 10% of cases, the common peroneal component of the sciatic nerve can pass through the division in the piriformis [16].
  • The most consistent internal rotators of the hip joint are the gluteus medius and tensor fascia latae muscles [16].
  • Lateral-sided hip pain is a conglomerate of conditions including snapping hip, trochanteric bursitis, and gluteal tendinopathy lumped together as greater trochanteric pain syndrome (GTPS) [20].
  • The largest bursa in the lateral hip is found between the gluteus maximus muscle and the gluteus medius tendon, which is located directly lateral to the greater trochanter [20].
  • The muscular sheaths and tendinous attachments of the gluteus maximus, ITB, tensor fascia lata, gluteus medius, and gluteus minimus contribute to a complex local environment susceptible to overuse injuries, direct trauma, and gait alterations [20].

Neurovascular Anatomy

  • The medial femoral circumflex artery is the main blood supply to the femoral head in adults [18].
  • The lateral femoral circumflex artery gives rise to the anterior aspect of the extracapsular arterial ring [18].
  • The superior and inferior gluteal arteries also contribute branches to the extracapsular arterial ring [18].
  • The ascending cervical arteries originate from the extracapsular arterial ring and are divided into lateral, medial, posterior, and anterior groups [18].
  • The lateral group of ascending branches is the main blood supply to the femoral head [18].
  • The lateral epiphyseal artery penetrates the femoral head and is believed to be the dominant blood supply to the femoral head from this system [18].
  • Fractures that disrupt the ascending blood flow to the lateral epiphyseal vessel have an increased risk of osteonecrosis [18].
  • The artery of the ligamentum teres arises from either the obturator or medial femoral circumflex artery and does not provide sufficient blood supply to maintain the viability of the femoral head [18].
  • The common femoral artery arises from the external iliac artery as it passes underneath the inguinal ligament [21].
  • The common femoral artery passes anterior and medial to the hip capsule [21].
  • The common femoral vessels are the most commonly reported extrapelvic vascular structures injured during total hip arthroplasty [21].
  • The most common mechanism of injury to the common femoral vessels is errant retractor placement anterior to the acetabulum [21].
  • The profundus or deep femoral artery arises from the lateral aspect of the common femoral artery approximately 3.5 cm below the inguinal ligament [21].
  • The lateral circumflex artery arises from the lateral side of the proximal profundus femoris artery [21].
  • The medial circumflex artery most commonly comes from the posteromedial profundus femoris artery but may also come directly from the femoral artery [21].
  • The superior gluteal vessels are branches of the posterior division of the internal iliac artery and are closest to the hip as they exit from the sciatic notch [21].
  • Superior gluteal artery injury can occur with the placement of screws in the region of the sciatic notch [21].
  • The inferior gluteal vessels and internal vessels are branches of the anterior division of the internal iliac artery and exit the pelvis between the piriformis and coccygeus muscles [21].
  • The inferior gluteal vessels can be injured by screws in the posterior column that are at least 5 mm past the bony margin [21].
  • The lateral femoral cutaneous nerve is at risk when using the anterior portal for hip arthroscopy, with less risk associated with the midanterior portal [3].
  • The sciatic nerve is the main structure at risk with the posterior (Southern) approach to the hip [18].
  • The superior gluteal nerve can be damaged if the intermuscular plane of the anterior lateral (Watson Jones) approach is extended to the iliac crest [18].

Pathophysiology

  • Femoroacetabular impingement (FAI) is recognized as a common cause of hip dysfunction and secondary osteoarthritis [3].
  • In FAI, distinct structural abnormalities produce repetitive impingement between the acetabulum and the femoral head-neck junction [3].
  • Three types of FAI are recognized: cam, pincer, and combined cam/pincer [3].
  • Cam impingement involves femoral-based abnormalities such as an aspherical femoral head and reduced head-neck offset, resulting in repetitive abutment of the acetabular rim and femoral head-neck junction [3].
  • Pincer impingement involves acetabular-based disorders such as acetabular retroversion, global overcoverage, and acetabular protrusio, creating abnormal abutment of the acetabular rim and femoral head-neck junction [3].
  • Impingement abnormalities can cause labral tears, degeneration, or ossification [3].
  • Impingement abnormalities can cause acetabular cartilage delamination [3].
  • Impingement abnormalities can cause secondary osteoarthritis [3].
  • The hip is a complex multiaxial joint capable of producing large forces and moving the thigh through large ranges of motion [1].
  • Any alteration to joint morphology or function can place the hip at risk for pathology [1].
  • The anatomic structures of the hip and pelvis are described in layers: Layer 1 is osseous morphology, Layer 2 is soft tissue including labrum, capsule, and ligaments, Layer 3 is the contractile layer including adductors, abductors, flexors/extensors, and rotators, and Layer 4 is neurovascular structures [12].
  • Patients with extra-articular hip pathology can have concomitant femoroacetabular impingement (FAI) or intra-articular hip pathology [12].
  • DDH is a gradually progressive disorder associated with distinct anatomic changes, many of which are initially reversible [13].
  • In unstable hips at birth, the posterosuperior rim of the acetabulum loses its sharp margin and becomes flattened and thickened in the area over which the femoral head slides [13].
  • A ridge of thickened articular cartilage called the neolimbus arises along the posterosuperior acetabular wall as the head rides in and out of the socket [13].
  • In dislocated hips, the fatty tissue known as the pulvinar thickens in the depths of the acetabulum and may impede reduction [13].
  • The ligamentum teres elongates and thickens in dislocated hips, taking up valuable space within the acetabulum [13].
  • The transverse acetabular ligament is often hypertrophic in dislocated hips and may impede reduction [13].
  • The inferior capsule of the hip assumes an hourglass shape in dislocated hips, eventually presenting an opening smaller in diameter than the femoral head [13].
  • The iliopsoas tendon is pulled tight across the capsular isthmus, contributing to narrowing and acting as a barrier to closed reduction [13].
  • Excessive pressure on the cartilaginous upper femur can cause a loss of vascular perfusion, resulting in necrosis of the chondrocytes [11].
  • Muscle imbalance can significantly affect the growth and morphology of the upper femur, with excessive adductor pull or inadequate abductor muscle function resulting in a valgus deformity [11].

Investigations

Clinical Examination

  • A thorough understanding of normal anatomy and biomechanics is necessary to identify pathology and determine the appropriate course of treatment for hip conditions [1].
  • A comprehensive clinical examination is required to determine a differential diagnosis because many hip conditions present with similar symptoms [1].
  • A thorough history is essential to differentiating between common causes of hip pain [1].
  • Clinical examination tests and imaging findings should be used to confirm a suspected clinical diagnosis [1].

Radiography

  • Conventional radiographs remain critical in the initial imaging evaluation of the hip [2].
  • Radiographs can be used to diagnose fractures, developmental dysplasia of the hip (DDH), femoroacetabular impingement (FAI), and osteoarthritis [2].
  • A complete hip series usually consists of an anterior-posterior (AP) pelvis, a centered AP hip, a lateral view (frog-leg, cross-table, Dunn 45° or 90°), and a false-profile (Lequesne) view [2].
  • The Dunn 45° view may be more accurate for determining the alpha angle measurement than CT or MRI [2].
  • The alpha angle is used to assess femoral head-neck junction morphology, with normal values generally considered less than 50°–55° [2].
  • The Tönnis angle is defined by the angle of the acetabular sourcil and a line parallel to the transverse pelvis axis, with values between 0° and 10° considered normal [2].
  • The lateral center-edge angle (Wiberg) is the angle between a line from the center of the femoral head perpendicular to the transverse pelvis axis and a second line from the center of the femoral head to the superolateral most point of the acetabulum [2].
  • Center-edge angles of 20°–40° are considered normal, while angles from 20° to 25° are considered borderline [2].
  • The femoral head extrusion index is defined by the length of the femoral head that lies beyond the acetabulum as a percentage of the total horizontal width of the femoral head [2].
  • Femoral head extrusion index values greater than 25% are considered abnormal [2].
  • Coxa profunda is diagnosed when the fossa line touches or is medial to the ilioischial line on an AP pelvis radiograph [2].
  • The "crossover" sign on an AP pelvis radiograph indicates acetabular retroversion related to lateralization of the anterior acetabular wall relative to the posterior acetabular wall [2].
  • Pelvic tilt or rotation may lead to false-positive and false-negative "crossover" signs on AP pelvis radiographs [2].
  • For neutral pelvic tilt on an AP pelvis radiograph, the sacrococcygeal joint should be between 3 and 5 cm above the superior border of the symphysis pubis [2].
  • Radiographs can serially assess hardware positioning and evaluate symptomatic hardware related to open reduction and internal fixation and total hip arthroplasty [2].
  • The AP pelvis view is used to assess acetabular anatomy, including version, acetabular coverage, and femoral head sphericity [3].
  • Various lateral views, most commonly the 45° Dunn view and frog-leg lateral, can be used to assess femoral head sphericity and head-neck offset [3].

Magnetic Resonance Imaging (MRI)

  • MRI is the modality of choice for patients suspected of soft tissue or intra-articular pathology, given its superior sensitivity and specificity [25].
  • Conventional MRI is effective at identifying osteochondral injuries, musculotendinous pathologies, and inflammation [25].
  • Magnetic resonance arthrography (MRA) is more appropriate than conventional MRI to determine injuries to the labrochondral structures and the ligamentum teres [25].
  • MRA is useful for identifying the presence of loose bodies and synovial chondromatosis [25].
  • The utility of MRA in the accurate detection and staging of articular cartilage lesions is reduced, with sensitivity reported to be less than 50% compared with arthroscopic findings [25].
  • Recent advances in MRI techniques, such as delayed gadolinium-enhanced MR imaging and T2* mapping, allow for a more in-depth analysis of the structure of articular cartilage [25].
  • Delayed gadolinium-enhanced MR imaging and T2* mapping were effective at detecting early changes to the articular cartilage surfaces of patients with hip dysplasia and femoroacetabular impingement [25].
  • MRI provides information regarding the integrity of the acetabular labrum and articular cartilage [3].
  • MRI can assess the anatomy of the proximal femur as well as the version of the acetabulum and femur [3].
  • The sensitivity of MRI to acetabular rim chondral lesions is limited [3].
  • Noncontrast MRI at 3T is generally adequate for diagnosing intra-articular pathology [28].
  • If 3T imaging is unavailable, MRA can be considered at 1.5T for increased diagnostic accuracy [28].
  • A prospective study found similar accuracies between noncontrast 3T MRI and 1.5T MRA in femoroacetabular impingement [26].
  • In a retrospective study evaluating noncontrast 3T MRI versus hip arthroscopy, the accuracy for labral tears was 98% and for acetabular cartilage lesions was 90% [26].
  • MRI is helpful in identifying femoral neck stress fracture in athletes and predicting patients that may require surgical intervention [28].
  • MRI is useful for assessing complications of conventional and resurfacing hip arthroplasties, particularly those with metal-on-metal bearing systems [28].
  • Major MRI findings that help predict histologic ALVAL scores include synovial thickening, synovitis, synovial volume, abductor disruption, and soft-tissue edema [28].
  • MRI is more sensitive than bone scan for ruling out occult nondisplaced stress fractures if the injury is less than 24 hours old [22].

Computed Tomography (CT)

  • CT scans are effective for examining cortical and cancellous bone and can be used to create three-dimensional reconstructions of the hip for use in surgical planning [25].
  • Measurements of femoral head coverage and acetabular and femoral impingement can be performed reliably using CT images [25].
  • Low-dose CT with three-dimensional reformats is particularly useful in surgical planning of complex or borderline deformities [3].
  • CT overcomes the limitations of radiography by providing three-dimensional assessment of bony morphology and, to some degree, assessment of soft-tissue abnormalities [10].
  • Combined with arthrography, CT can evaluate chondrolabral abnormalities, specifically in patients with contraindications to MRI [10].
  • CT is helpful in fracture evaluation, particularly in the setting of negative radiographs or for further defining fracture morphology in patients requiring surgical reduction [10].
  • The multiplanar and 3D capabilities of CT make it an invaluable tool for assessing bone morphology, but at higher cost and radiation dose [28].
  • 3D volume renderings are useful to aid in preoperative planning in FAI and subspine impingement [28].
  • CT is performed after reduction of hip dislocations to evaluate for associated acetabular and/or femoral head fracture and loose bodies in the joint [22].
  • CT is used to evaluate the location and size of femoral head fracture fragments and rule out associated acetabular fracture [22].

Ultrasonography

  • Ultrasonography provides real-time dynamic assessment of the hip and is useful in diagnosing soft-tissue abnormalities about the hip joint [10].
  • Ultrasonography is particularly useful in providing real-time guidance during diagnostic and therapeutic procedures [10].
  • Ultrasonography can be an effective modality to identify musculotendinous disruptions, effusions associated with intra-articular pathology, or inflammatory conditions, such as bursitis [25].
  • Ultrasonography is increasingly used for targeted injections into muscles, tendons, or intra-articularly around the hip for use with corticosteroids or biologic treatments [25].
  • Ultrasonography allows bedside evaluation of the hip and can be used to guide interventions in the office setting [28].
  • Ultrasonography provides dynamic assessment of structures such as tendon and nerve subluxation [29].
  • Higher frequency ultrasonographic beams can provide higher resolution images of superficial structures such as tendons and ligaments [29].
  • Ultrasonography cannot image inside bone because bone cortex reflects almost all sound waves [29].
  • Internal joint structures are not well visualized by ultrasonography unless they are in a superficial location [29].
  • Image quality and interpretation of ultrasonography depend on the experience of the ultrasonography technician and the radiologist [29].
  • The utility of ultrasonography in evaluating the adult hip is limited, although it is a valuable tool to examine pediatric hip conditions [25].

References

[1] Orthopaedic Knowledge Update 13 Ebook Without Multimedia. Anatomy and Biomechanics, Evaluation, Clinical Examination, and Imaging of the Hip > Summary.

[2] Orthopaedic Knowledge Update Sports Medicine 6. Imaging of the Hip > Radiography.

[3] Aaos Comprehensive Orthopaedic Review 3. Nonarthroplasty Surgical Treatment of the Hip > I. Femoroacetabular Impingement.

[5] Miller S Review Of Orthopaedics. Genetics of musculoskeletal conditions and abnormalities are summarized in Table 1.27 > 2. Arthrology > Hip (Fig. 2.49).

[8] Orthopaedic Knowledge Update 13 Ebook Without Multimedia. Anatomy and Biomechanics, Evaluation, Clinical Examination, and Imaging of the Hip > Osseous and Ligamentous Anatomy.

[9] Aaos Comprehensive Orthopaedic Review 3. Surgical Anatomy of the Hip > IV. Hip Capsule and Ligaments.

[10] Orthopaedic Knowledge Update Sports Medicine 6. Imaging of the Hip > Introduction.

[11] Tachdjian S Pediatric Orthopaedics From The Texas Scottish Rite Hospital For Children E Book. Associated Conditions > Pathophysiology.

[12] Orthopaedic Knowledge Update Sports Medicine 6. Core Muscle Injury/Athletic Pubalgia and Groin Pathology > Introduction.

[13] Tachdjian S Pediatric Orthopaedics From The Texas Scottish Rite Hospital For Children E Book. Hip Development With Developmental Dysplasia of the Hip.

[14] Miller S Review Of Orthopaedics. SECTION 16 PATELLAR TRACKING IN TOTAL KNEE ARTHROPLASTY > LOWER EXTREMITY.

[16] Aaos Comprehensive Orthopaedic Review 3. Surgical Anatomy of the Hip > V. Hip Joint Muscles.

[18] Aaos Comprehensive Orthopaedic Review 3. Fractures of the Hip > I. General Considerations.

[20] Orthopaedic Knowledge Update 13 Ebook Without Multimedia. Muscular, Neurovascular, and Soft-­Tissue Conditions of the Hip > Muscular Conditions About the Hip > Hip Bursitis.

[21] Aaos Comprehensive Orthopaedic Review 3. Surgical Anatomy of the Hip > VI. Neurovascular Structures Surrounding the Hip.

[22] Miller S Review Of Orthopaedics. SECTION 16 PATELLAR TRACKING IN TOTAL KNEE ARTHROPLASTY > YOUNG ADULT PROXIMAL FEMUR INJURIES.

[25] Orthopaedic Knowledge Update 13 Ebook Without Multimedia. Anatomy and Biomechanics, Evaluation, Clinical Examination, and Imaging of the Hip > Imaging.

[26] Orthopaedic Knowledge Update Sports Medicine 6. Imaging of the Hip > Annotated References.

[28] Orthopaedic Knowledge Update Sports Medicine 6. Imaging of the Hip > Summary.

[29] Aaos Comprehensive Orthopaedic Review 3. Musculoskeletal Imaging* > IV. Ultrasonography.

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