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Patients › Hip

髋关节表面置换术

Updated Sep 2026
Illustration: hip

本页面由机器翻译,尚未经临床医生审核。英文版本为权威版本。

为何建议进行此手术

在此手术中,您受损的髋关节表面会被金属覆盖,而非被切除并替换,因此您自身的骨骼得以更多地保留在原位。

髋关节表面置换术通常适用于较年轻、活动量大的骨关节炎患者,尤其是 55 岁以下的男性。对于退行性或长期存在的问题,我们通常首先尝试非手术治疗,例如改变活动方式或进行物理治疗,并在这些方法未能带来足够改善时考虑手术。该手术旨在缓解疼痛,让您能够保持活跃。大多数接受此手术的人会继续参与体育运动,其中 87% 在术后继续从事体育活动。我们将与您讨论该手术是否适合您,并共同做出决定。

手术前

在手术前,我们会利用您的髋部X光片进行仔细规划,有时还会使用磁共振成像(MRI,一种显示软组织的扫描)或超声检查。这些扫描有助于我们选择合适的植入物尺寸和位置。在手术前的几天里,我们会向您提供明确的指示。您需要提前七小时停止进食和饮水。我们要求七小时而非更短的时间,以便如果手术室排班提前结束,您的手术可以提前进行。某些药物可能需要暂停使用,我们会告知您具体是哪些药物以及何时暂停。如果您有其他健康状况,可能需要进行血液检查或由麻醉师(负责手术期间您护理的专家)进行评估。当天,请携带您目前用药的清单,穿着舒适的衣物,并安排有人开车送您回家。

手术当日

您将抵达医院的手术入院病区,在此办理入院手续并进行术前准备。您将与麻醉师(负责在手术期间照看您的专科医生)见面。该手术在全身麻醉下进行。有时会追加区域神经阻滞以缓解术后疼痛;麻醉师将在当日就此与您讨论。随后,您将被带入手术室进行手术。

您将在复苏区苏醒,期间护士会监测您的状况,直至麻醉作用消退。待您的生命体征稳定后,根据手术类型及您的恢复情况,您将被转入病房或直接回家。

手术内容

髋关节表面置换术与全髋关节置换术不同。外科医生不会切除并替换股骨顶端,而是将磨损的股骨头修整成光滑的穹顶状,并在其上覆盖金属帽。骨盆中磨损的髋臼也会安装一个薄金属壳。这两个部件均固定在您自身的骨骼上,因此髋关节的大部分结构保持原位。

为了到达髋关节,外科医生会在大腿上段外侧做一个切口,将肌肉向两侧牵开而非切断。清除受损的软骨(骨骼末端的平滑衬里),并将骨骼修整以适配金属部件。髋臼内的金属壳被牢固地压入就位,使其能够自行固定。股骨上的金属帽使用骨水泥固定,这是一种硬化后能将部件锚定在骨骼上的材料。外科医生在关闭切口(缝合并包扎)前,会检查部件是否配合良好且运动顺畅。

该手术旨在尽可能多地保留您自身的骨骼。由于未切除股骨顶端,未来数年您的身体可依赖的骨骼量更多。这也是该手术适合年轻活跃人群的原因之一,如本页面前述内容所述。

在手术当天之前,外科医生会根据您的影像检查和体格检查结果,向您详细说明髋关节手术方案。如果对手术的任何环节有疑问,请提出。在签署知情同意书之前了解每个步骤是有帮助的。

术后

您将在恢复区苏醒,随着麻醉药效消退,护士会密切观察您的状况。护士会定期检查您的疼痛情况,并给予药物以确保您的舒适。您的髋部切口处会覆盖敷料,以固定敷料并保持伤口清洁。敷料通常保留约10天;除非我们告知您,否则请勿在此之前自行拆除。我们会在复诊时为您更换或拆除敷料。在术后第一天,通常在手术后的几小时内,物理治疗师会协助您起身,并使用助行器迈出第一步。回家后,最初的24小时内应有人陪伴您。您的医疗团队会告知您是当天出院还是在医院过夜。

恢复

术后最初几天,您的髋部会感到疼痛,切口周围会出现肿胀。这是正常现象,并会逐渐消退。止痛药可在早期阶段帮助您保持舒适,随着每天过去,大多数人所需的药量会减少。休息、温和的活动以及遵循物理治疗师的指导都有助于缓解不适。

物理治疗师将从一开始指导您的恢复。您将进行简单的锻炼以增强力量并保持髋部活动。起初,您需要借助助行器行走,然后随着腿部感觉稳固,逐渐增加腿部承重。在家中,您可以四处走动并进行轻微的日常生活活动,但在外科医生告知安全之前,您需要避免深度弯曲、扭转或提重物。起初仰卧通常更舒适,有些人会在膝盖之间放一个枕头。

恢复的里程碑以事件而非日期来衡量。一旦肿胀消退,活动会感觉更轻松。一旦外科医生允许您驾驶,您即可重返道路;我们单独的驾驶指南解释了适用的规则。当外科医生对您的力量和活动能力感到满意时,您可以分阶段返回工作和运动。

恢复因人而异。您的时间线可能有所不同,您的外科医生和物理治疗师将在每个步骤中为您提供指导。

可能出现的并发症

大多数患者恢复良好,但偶尔也会出现问题。您的外科医生和医疗团队会密切监测您的状况,以便尽早发现任何问题。

需要再次手术的最常见原因是股骨上的金属帽出现问题。有时金属帽会松动,或者其下方的骨骼会发生骨折。您可能会注意到腹股沟或大腿出现与既往关节炎疼痛不同的深部搏动性疼痛,或者在绊倒后突然出现的疼痛。髋关节可能会感觉不稳定或出现研磨感。如果发生这种情况,请联系诊所。您可能需要进行影像学检查,有时需要更换金属帽。

部分患者会对植入物的金属表面产生反应。随着时间的推移,微小的金属颗粒可能从轴承上磨损下来,并进入周围组织或您的血液循环中。这可能导致髋关节周围的疼痛、肿胀或积液,或在活动时产生摩擦感。如果您注意到这些变化,请在下次复诊时告知您的外科医生,以便其安排血液检查和影像学检查。

与全髋关节置换术相比,此类手术早期并发症的发生率往往更高,且与外科医生对该技术的经验相关。在此方面,外科医生的培训和手术量至关重要,在咨询时询问相关情况是合理的。

如果确实需要更换金属帽,通常通过安装新的金属帽或转换为全髋关节置换术来完成。该手术后的行走和日常功能通常与直接进行全髋关节置换术后的情况相似。

此手术保留了您自身的骨骼,这在将来如果需要再次手术时非常重要。保留在股骨和骨盆中的骨骼为外科医生提供了更多的操作空间。

如果您想了解具体数据,本页上的并发症表格列出了典型的发生率。

何时联系我们

大多数问题都会早期显现,因此了解需要警惕的症状至关重要。如果您出现发热,切口周围皮肤发红加重或开始渗出液体,或疼痛持续加剧而非缓解,请致电我们。如果您出现突发的剧烈疼痛、小腿肿胀或呼吸急促,请立即前往急诊。如果您出现腿部感觉丧失或无法活动,也请立即前往急诊。如果您注意到腹股沟疼痛(如前文所述的深部搏动性疼痛),或髋部周围出现肿胀或积液,请联系诊所。我们宁愿接到您的电话,也不愿让您独自在家担忧。


Evidence & references

This is the clinical evidence summary written for health professionals. It is technical, and it lists the research this page was built from. You do not need to read it to understand your treatment or to make a decision about it.

Anatomy & Pathophysiology

Bony Anatomy

  • The hip is a multiaxial joint formed by the articulation between the pelvis and femur, connecting the axial skeleton and the lower extremity [7].
  • The hemipelvis comprises three bones: the ilium, ischium, and pubis, which unite at the triradiate cartilage within the concave acetabulum [7].
  • The acetabulum comprises an articular crescent-moon-shaped lunate surface and a nonarticular central fossa that serves as the attachment point for the ligamentum teres [7].
  • The acetabulum is incomplete inferiorly, forming a notch through which vital blood vessels and nerves pass to supply the joint [7].
  • The femoral head forms two-thirds of a sphere, with a small depression at its center from which the ligamentum teres extends to connect to the acetabular notch [7].
  • The neck-shaft angle of the femur averages 125° [7].
  • Normal version, defined as the head-neck angle in the frontal plane, averages 15 to 20° [7].
  • The mean femoral neck-shaft angle in the adult is 130° ± 7° [13].
  • The mean anteversion of the femoral neck is 10° ± 7° [13].
  • The two prime trabecular groups of the proximal femur are the principal tensile group and the principal compressive group [13].
  • The weakest area in the femoral neck is located in the Ward triangle [13].
  • The calcar femorale is a medial area of dense trabecular bone that transfers stress from the femoral shaft to the inferior portion of the femoral neck [13].

Soft Tissue Anatomy

  • The acetabular labrum is a fibrocartilaginous ring attached to the rim of the acetabulum that extends the articulating surface area and increases femoral head coverage [7].
  • The labrum is triangular in cross section, which contributes to its ability to create a pressurized seal of the central compartment of the hip during loading [7].
  • Only the external one-third of the labrum contains blood vessels, leaving the majority of the structure avascular and limiting its healing ability following injury [7].
  • The labrum is highly innervated, with the presence of both mechanoreceptors and nociceptors [7].
  • The transverse acetabular ligament serves as the continuation of the labrum in the area of the inferior acetabular notch, connecting the anterior and posterior lunate surfaces [7].
  • The hip is surrounded by a dense fibrous capsule extending from the periphery of the acetabulum to the intertrochanteric line of the femoral neck [7].
  • The iliofemoral ligament is Y-shaped, with the medial portion connecting the anterior inferior iliac spine to the anterior intertrochanteric line and the lateral portion attaching to the anterior greater trochanter [7].
  • The iliofemoral ligament functions to limit external rotation, while in isolation, the lateral arm limits extension of the joint [7].
  • The ischiofemoral ligament extends from the ischial margin of the acetabulum to the greater trochanter of the femur and restricts internal rotation motion [7].
  • The pubofemoral ligament extends from the obturator crest of the pubic bone to the femoral neck and acts to limit abduction of the joint [7].
  • Deep fibers from the iliofemoral, ischiofemoral, and pubofemoral ligaments merge to form the zona orbicularis, which circumvents the femoral neck [7].
  • The ligamentum teres arises from the apex of the cotyloid notch and attaches to the fovea of the femoral head [4].
  • The ligamentum teres transmits an arterial branch of the posterior division of the obturator artery to the femoral head, which is less significant in adults [4].
  • The hip joint capsule extends anteriorly to the intertrochanteric crest but posteriorly only partially across the femoral neck, leaving the basicervical and intertrochanteric crest regions extracapsular [4].
  • The iliofemoral ligament becomes taut in full extension, preventing anterior dislocation and hyperextension of the hip [8].
  • The twisted orientation of the hip ligaments provides a screw mechanism for the hip in full extension [8].

Vascular Anatomy

  • In adulthood, the major blood supply to the femoral head is from the medial femoral circumflex and lateral epiphyseal arteries [16].
  • The medial femoral circumflex artery is the main blood supply to the femoral head and terminates in the posterior aspect of the extracapsular arterial ring [13].
  • The lateral group of ascending branches from the extracapsular arterial ring is the main blood supply to the femoral head [13].
  • The lateral epiphyseal artery penetrates the femoral head and is believed to be the dominant blood supply to the femoral head from the ascending cervical system [13].
  • The artery of the ligamentum teres arises from either the obturator or medial femoral circumflex artery and does not provide sufficient blood supply to maintain the viability of the femoral head [13].
  • Fractures that disrupt the ascending blood flow to the lateral epiphyseal vessel have an increased risk of osteonecrosis [13].

Pathophysiology

  • Femoroacetabular impingement (FAI) is recognized as a common cause of hip dysfunction and secondary osteoarthritis [3].
  • In FAI, distinct structural abnormalities produce repetitive impingement between the acetabulum and the femoral head-neck junction [3].
  • Cam impingement involves femoral-based abnormalities such as an aspherical femoral head and reduced head-neck offset, resulting in repetitive abutment of the acetabular rim and femoral head-neck junction [3].
  • Pincer impingement involves acetabular-based disorders such as acetabular retroversion, global overcoverage, and acetabular protrusio, creating abnormal abutment of the acetabular rim and femoral head-neck junction [3].
  • Impingement abnormalities can cause labral tears, degeneration, or ossification [3].
  • Impingement abnormalities can cause acetabular cartilage delamination [3].
  • Impingement abnormalities can cause secondary osteoarthritis [3].
  • Hip microinstability refers to femoral head micromotion within the acetabulum, which is a prolonged phenomenon that leads to cartilage damage and eventually osteoarthritis of the hip [17].
  • Acetabular hip dysplasia can contribute to hip instability because of a shallow acetabular component [17].
  • Developmental dysplasia of the hip (DDH) is a gradually progressive disorder associated with distinct anatomic changes, many of which are initially reversible [10].
  • In unstable hips at birth, the posterosuperior rim of the acetabulum loses its sharp margin and becomes flattened and thickened in the area over which the femoral head slides [10].
  • A ridge of thickened articular cartilage called the neolimbus arises along the posterosuperior acetabular wall in dislocatable hips [10].
  • In dislocated hips, the fatty tissue known as the pulvinar thickens in the depths of the acetabulum and may impede reduction [10].
  • In dislocated hips, the ligamentum teres elongates and thickens, taking up valuable space within the acetabulum [10].
  • In dislocated hips, the inferior capsule assumes an hourglass shape, presenting an opening smaller in diameter than the femoral head [10].
  • The iliopsoas tendon is pulled tight across the capsular isthmus in dislocated hips, contributing to narrowing and acting as a barrier to closed reduction [10].
  • Femoral neck fractures are considered intracapsular fractures at higher risk of nonunion due to the absence of a periosteal or extraosseous blood supply [13].
  • Intertrochanteric fractures are considered extracapsular fractures where callus formation is common and nonunion is rare due to the presence of an abundant blood supply [13].
  • Periprosthetic femoral fractures associated with hip resurfacing are categorized into Type A (biomechanical), Type B (acute postnecrotic), and Type C (chronic biomechanical) patterns [34].
  • Type A periprosthetic fractures after hip resurfacing occur at an average of 41 days postoperatively and are characterized by acute fracture changes without signs of osteonecrosis [34].
  • Type B periprosthetic fractures after hip resurfacing occur at an average of 149 days postoperatively and are all associated with osteonecrosis [34].
  • Type C periprosthetic fractures after hip resurfacing occur at an average of 179 days postoperatively and are characterized by evidence of refracture or pseudoarthrosis through a previous fracture [34].
  • The majority of periprosthetic femoral fractures after hip resurfacing occur inside the bounds of the edge of the femoral head component [34].
  • Acute biomechanical periprosthetic fractures after hip resurfacing are located exclusively outside of the component and in the neck [34].

Investigations

Clinical Examination

  • A thorough history is essential to differentiate between common causes of hip pain [1].
  • A comprehensive clinical examination is required to determine a differential diagnosis because many hip conditions present with similar symptoms [1].
  • Clinical examination tests and imaging findings should be used to confirm a suspected clinical diagnosis [1].
  • Patients with symptomatic femoroacetabular impingement (FAI) frequently present with activity-related groin pain exacerbated by hip flexion activities [3].
  • Patients with symptomatic FAI may experience difficulty with prolonged sitting, walking, running, or pivoting [3].
  • The onset of symptoms in FAI is often insidious or follows minor trauma [3].
  • Patients with FAI exhibit restricted hip internal rotation in 90° of flexion [3].
  • The impingement test (flexion, adduction, internal rotation) elicits pain in patients with FAI but is not specific for the condition [3].
  • The impingement test involves hip flexion to 90 degrees followed by hip adduction and internal rotation to yield a pain response [30].
  • The roll test involves rolling the leg into internal and external rotation while the patient is supine, with a positive result indicated by stiffness or grabbing [30].
  • The Stinchfield test involves an active straight-leg raise of approximately 20 cm against mild resistance, with a positive result indicated by pain in the anterior hip [30].
  • The Patrick test involves positioning the leg in a figure-of-four position, with pain in the anterior or posterior hip region indicating a positive result [30].
  • Pain located over the posterior pelvis during the Patrick test indicates referred pain from L5 to S1 facets or the sacroiliac joint rather than the hip joint [30].

Radiography

  • Conventional radiographs remain critical in the initial imaging evaluation of the hip [2].
  • Radiographs can be used to diagnose fractures, developmental dysplasia of the hip (DDH), femoroacetabular impingement (FAI), and osteoarthritis [2].
  • A complete hip series usually consists of an anterior-posterior (AP) pelvis, a centered AP hip, a lateral view (frog-leg, cross-table, Dunn 45° or 90°), and a false-profile (Lequesne) view [2].
  • Osteoarthritis of the hip can be categorized using the Kellgren-Lawrence classification, which is a 4-point grading system classified into doubtful, mild, moderate, and severe [2].
  • Osteoarthritis of the hip can be categorized using the Tönnis classification, which is a 3-point grading system categorized into mild, moderate, and severe [2].
  • Radiographic and clinical severity of osteoarthritis do not necessarily correlate, particularly if radiographs are non-weight-bearing or false-profile views are not included [2].
  • Acetabular morphology is assessed on AP pelvis radiographs to evaluate acetabular overcoverage and undercoverage [2].
  • The femoral head-neck junction morphology is often assessed using the alpha angle [2].
  • Some studies have shown that radiographs, in particular the Dunn 45° view, may be more accurate for determining the alpha angle measurement than CT or MRI [2].
  • The AP pelvis radiograph should be performed with the lower extremities in approximately 15° of internal rotation and centered over the pelvis [2].
  • Coxa profunda is diagnosed when the acetabular fossa line touches or is medial to the ilioischial line [2].
  • The femoral head extrusion index is defined by the length of the femoral head that lies beyond the acetabulum as a percentage of the total horizontal width of the femoral head [2].
  • Femoral head extrusion index values greater than 25% are considered abnormal [2].
  • The Tönnis angle is defined by the angle of the acetabular sourcil and a line parallel to the transverse pelvis axis [2].
  • Tönnis angles between 0° and 10° are considered normal [2].
  • The lateral center-edge angle of Wiberg is the angle between a line from the center of the femoral head perpendicular to the transverse pelvis axis and a second line from the center of the femoral head to the superolateral most point of the acetabulum [2].
  • Center-edge angles of 20°–40° are considered normal, while angles from 20° to 25° are considered borderline [2].
  • A normal alpha angle is less than 50°–55° [2].
  • An abnormal alpha angle of 70° indicates femoroacetabular impingement [2].
  • The “crossover” sign on an AP pelvis radiograph indicates acetabular retroversion related to lateralization of the anterior acetabular wall relative to the posterior acetabular wall [2].
  • Pelvic tilt or rotation may lead to false-positive and false-negative “crossover” signs [2].
  • For neutral pelvic tilt, the sacrococcygeal joint should be between 3 and 5 cm above the superior border of the symphysis pubis [2].
  • An increased Tönnis angle of 19° and an increased femoral head extrusion index of 28% are indicative of developmental dysplasia of the hip [2].
  • A reduced anterior center edge angle of 17° on a false profile radiograph is indicative of developmental dysplasia of the hip [2].
  • Radiographs remain integral to the assessment of fractures and can be supplemented with CT to investigate suspected occult fractures, define fracture morphology, and assist in preoperative planning [2].
  • Radiographs can serially assess hardware positioning and evaluate symptomatic hardware related to open reduction and internal fixation and total hip arthroplasty [2].
  • Standard AP radiographs of the hip and pelvis are obtained to examine bony architecture, check for evidence of joint space narrowing or changes to bone quality, and quantify femoral head coverage [21].
  • The Dunn view and frog leg view are appropriate to measure the alpha angle to determine the presence of impingement [21].
  • Plain radiographs will identify the fracture in the majority of cases for femoral neck fractures [28].
  • Anteroposterior (AP) and lateral radiographs are required for the evaluation of femoral neck fractures [28].
  • In equivocal cases, the lateral radiograph can help determine whether a femoral neck fracture is displaced, which is usually essential to determine the choice of treatment [28].
  • Full-length AP and lateral femur films are standard for femoral neck fracture evaluation [28].

Magnetic Resonance Imaging

  • MRI is the modality of choice for patients suspected of soft tissue or intra-articular pathology, given its superior sensitivity and specificity [21].
  • Conventional MRI is effective at identifying osteochondral injuries, musculotendinous pathologies, and inflammation [21].
  • Magnetic resonance arthrography (MRA) is more appropriate than conventional MRI to determine injuries to the labrochondral structures and the ligamentum teres [21].
  • MRA is used to identify the presence of loose bodies and synovial chondromatosis [21].
  • The utility of MRA in the accurate detection and staging of articular cartilage lesions is reduced, with sensitivity reported to be less than 50% compared with arthroscopic findings [21].
  • Recent advances in MRI imaging techniques, such as delayed gadolinium-enhanced MR imaging and T2* mapping, allow for a more in-depth analysis of the structure of articular cartilage [21].
  • Delayed gadolinium-enhanced MR imaging and T2* mapping were effective at detecting early changes to the articular cartilage surfaces of patients with hip dysplasia and femoroacetabular impingement [21].
  • MRI provides information regarding the integrity of the acetabular labrum and articular cartilage [3].
  • MRI can assess the anatomy of the proximal femur as well as the version of the acetabulum and femur [3].
  • The sensitivity of MRI to acetabular rim chondral lesions is limited [3].
  • MRI is used when osteonecrosis is suspected [30].
  • Gadolinium-enhanced MRI arthrogram is useful when labral pathology is suspected, especially when associated with FAI [30].
  • MRI may identify gluteus medius and gluteus minimus tears in patients with lateral hip pain and abductor weakness [30].
  • Noncontrast MRI at 3T is generally adequate for diagnosing intra-articular pathology [26].
  • If 3T imaging is unavailable, MRA can be considered at 1.5T for increased diagnostic accuracy [26].
  • MRI is helpful in identifying femoral neck stress fracture in athletes and predicting patients that may require surgical intervention [26].
  • MRI is helpful in assessing complications of conventional and resurfacing hip arthroplasties, particularly those with metal-on-metal bearing systems [26].
  • Major MRI findings that help predict histologic ALVAL scores include synovial thickening, synovitis, synovial volume, abductor disruption, and soft-tissue edema [26].
  • A prospective study found similar accuracies between noncontrast 3T MRI and 1.5T MRA in femoroacetabular impingement [24].
  • In a retrospective study evaluating noncontrast 3T MRI versus hip arthroscopy, accuracy for labral tears was 98% and for acetabular cartilage lesions was 90% [24].
  • MRI is the current additional imaging modality recommended where there is uncertainty about the presence of an intracapsular fracture [28].
  • MRI is more accurate than CT in detecting occult hip fractures in patients with normal radiographs [28].
  • MRI is more accurate than a bone scan in the early stages after injury for detecting occult hip fractures and has no radiation [28].
  • MRI will demonstrate soft tissue problems that may be causing hip pain in the absence of a fracture [28].
  • Positron emission tomography/computed tomography (PET/CT) at 6 weeks could detect recovery of vascularity and predict the risk of vascular necrosis [28].
  • Dynamic MRI–positive enhancement integral color mapping (PEICM) can estimate femoral head perfusion preoperatively [28].
  • In a study using dynamic MRI-PEICM, the nonunion rate was zero in the normal perfusion group, 6.7% in the reduced perfusion group, and 50% in the absent perfusion group [28].

Computed Tomography

  • CT scans are effective for examining cortical and cancellous bone [21].
  • CT can be used to create three-dimensional reconstructions of the hip for use in surgical planning [21].
  • Measurements of femoral head coverage and acetabular and femoral impingement can be performed reliably using CT images [21].
  • Low-dose CT with three-dimensional reformats is particularly useful in surgical planning of complex or borderline deformities [3].
  • CT overcomes the limitations of radiography by providing three-dimensional assessment of bony morphology and, to some degree, assessment of soft-tissue abnormalities [9].
  • Combined with arthrography, CT can evaluate chondrolabral abnormalities, specifically in patients with contraindications to MRI [9].
  • CT is helpful in fracture evaluation, particularly in the setting of negative radiographs or for further defining fracture morphology in patients requiring surgical reduction [9].
  • Three-dimensional CT with pelvic remodeling may be indicated for preoperative planning for reconstruction associated with dysplasia surgery, femoroacetabular impingement, posttraumatic arthritis, or other complex primary total hip arthroplasty [30].
  • CT scanning is a more accurate investigation than technetium bone scan for suspected fractures but exposes the patient to further radiation [28].
  • Multidetector CT scanning reported 100% specificity and sensitivity in the diagnosis of hip fracture in a series of 209 patients with negative plain radiographs [28].
  • In a series of 78 CT scans, CT scanning yielded a sensitivity of 86% and specificity of 98% for occult hip fractures [28].
  • The multiplanar and 3D capabilities of CT make it an invaluable tool for assessing bone morphology, but at higher cost and radiation dose [26].
  • 3D volume renderings are useful to aid in preoperative planning in FAI and subspine impingement [26].

Ultrasonography

  • Ultrasonography provides real-time dynamic assessment of the hip [9].
  • Ultrasonography is useful in diagnosing soft-tissue abnormalities about the hip joint and, to a lesser degree, within the hip joint itself [9].
  • Ultrasonography is particularly useful in providing real-time guidance during diagnostic and therapeutic procedures [9].
  • Although ultrasonography is a valuable tool to examine pediatric hip conditions, its utility in evaluating the adult hip is limited [21].
  • Ultrasonography can be an effective modality to identify musculotendinous disruptions, effusions associated with intra-articular pathology, or inflammatory conditions such as bursitis [21].
  • Ultrasonography is increasingly used for targeted injections into muscles, tendons, or intra-articularly around the hip for corticosteroids or biologic treatments [21].
  • Ultrasonography allows bedside evaluation of the hip and can be used to guide interventions in the office setting [26].

Treatment

  • Dislocation is a recognized complication following total hip replacement [33].
  • Metal-on-metal total hip arthroplasty has specific modes of failure [33].
  • Osteolysis is a current concept in orthopaedic management [33].
  • The direct anterior approach is a technique used for total hip arthroplasty [33].
  • Periprosthetic femoral fractures are a complication of total hip arthroplasty that require classification and treatment [33].
  • Acetabular bone loss is evaluated and managed in revision total hip arthroplasty [33].
  • Femoral bone loss is evaluated and managed in revision total hip arthroplasty [33].
  • Nonarthroplasty surgical options are available for hip disease in the young, active patient [33].
  • Osteonecrosis of the femoral head is evaluated and treated [33].

References

[1] Orthopaedic Knowledge Update 13 Ebook Without Multimedia. Anatomy and Biomechanics, Evaluation, Clinical Examination, and Imaging of the Hip > Summary.

[2] Orthopaedic Knowledge Update Sports Medicine 6. Imaging of the Hip > Radiography.

[3] Aaos Comprehensive Orthopaedic Review 3. Nonarthroplasty Surgical Treatment of the Hip > I. Femoroacetabular Impingement.

[4] Miller S Review Of Orthopaedics. SECTION 16 PATELLAR TRACKING IN TOTAL KNEE ARTHROPLASTY > 2. Arthrology > Hip (Fig. 2.49).

[7] Orthopaedic Knowledge Update 13 Ebook Without Multimedia. Anatomy and Biomechanics, Evaluation, Clinical Examination, and Imaging of the Hip > Osseous and Ligamentous Anatomy.

[8] Aaos Comprehensive Orthopaedic Review 3. Surgical Anatomy of the Hip > IV. Hip Capsule and Ligaments.

[9] Orthopaedic Knowledge Update Sports Medicine 6. Imaging of the Hip > Introduction.

[10] Tachdjian S Pediatric Orthopaedics From The Texas Scottish Rite Hospital For Children E Book. Hip Development With Developmental Dysplasia of the Hip.

[13] Aaos Comprehensive Orthopaedic Review 3. Fractures of the Hip > I. General Considerations.

[16] Aaos Comprehensive Orthopaedic Review 3. Surgical Anatomy of the Hip > VI. Neurovascular Structures Surrounding the Hip.

[17] Orthopaedic Knowledge Update Sports Medicine 6. Hip Microinstability > Introduction.

[21] Orthopaedic Knowledge Update 13 Ebook Without Multimedia. Anatomy and Biomechanics, Evaluation, Clinical Examination, and Imaging of the Hip > Imaging.

[24] Orthopaedic Knowledge Update Sports Medicine 6. Imaging of the Hip > Annotated References.

[26] Orthopaedic Knowledge Update Sports Medicine 6. Imaging of the Hip > Summary.

[28] Rockwood And Green S Fractures In Adults. 51: Hip Dislocations and Femoral Head Fractures > Imaging and Other Diagnostic Studies for Femoral Neck Fractures.

[30] Miller S Review Of Orthopaedics. SECTION 16 PATELLAR TRACKING IN TOTAL KNEE ARTHROPLASTY > SECTION 1 EVALUATION OF THE ADULT PATIENT WITH HIP PAIN.

[33] Miller S Review Of Orthopaedics. SECTION 16 PATELLAR TRACKING IN TOTAL KNEE ARTHROPLASTY > HIP.

[34] Rockwood And Green S Fractures In Adults. Mechanisms of Injury for Distal Femur Fractures > Classification of Periprosthetic Femur Fractures After Hip Resurfacing.

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