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Codo de golfista

Golfer’s elbow (medial epicondylitis) — causes, symptoms, and conservative treatment options.

Updated Sep 2026
Una ilustración dibujada a mano de una persona sin rostro en pleno swing de golf.
Codo de golfista: los tendones que flexionan la muñeca y los dedos se insertan en una protuberancia ósea en la parte interna del codo; el uso excesivo provoca dolor y degeneración en el punto de unión con el hueso. Kieran Hirpara 4.0

Esta página se tradujo automáticamente y todavía no la ha revisado un médico. La versión en inglés es la versión oficial.

Qué está sintiendo

El codo de golfista se caracteriza por dolor en la parte interna del codo, sobre una pequeña protuberancia ósea llamada epicóndilo medial. Los tendones que se insertan allí ayudan a agarrar objetos, a flexionar la muñeca y a girar el antebrazo. Cuando se irritan, el dolor suele extenderse hacia la parte superior del antebrazo.

Por lo general, el dolor aparece de forma gradual, sin que haya habido una lesión evidente. Tiende a empeorar con las actividades que lo provocan, como jugar al golf, lanzar objetos, jugar al tenis o realizar trabajos que requieran un agarre fuerte. Algunas personas lo notan sobre todo durante el movimiento de lanzamiento; otras lo sienten más después de realizar actividad física o al despertar por la mañana. El descanso puede aliviarlo temporalmente, pero el dolor suele reaparecer en cuanto se vuelve a realizar las mismas tareas.

Acciones cotidianas que someten a esos tendones a esfuerzo pueden resultar incómodas: levantar una bolsa pesada de la compra, cargar cargas de unos 20 kg o más en el trabajo, agarrar herramientas o girar un pomo de puerta pueden empeorar los síntomas. Es posible que note que su fuerza de agarre es menor que en el otro brazo. Algunas personas también experimentan hormigueo o sensibilidad a lo largo de la parte interna del codo, donde pasa un nervio cercano.

En la mayoría de los casos, se puede mover el codo y la muñeca dentro de su rango completo de movimiento. El dolor suele localizarse justo delante y debajo de la protuberancia ósea; a veces hay cierta hinchazón blanda en esa zona. Si los síntomas aparecieron repentinamente tras un golpe directo en el codo, o si no puede estirar completamente el brazo, eso indica un problema distinto que requiere evaluación inmediata.

El dolor en la parte interna del codo también puede deberse a otras causas, como irritación nerviosa, lesión ligamentosa o artritis en el cuello o en el codo. Por ello, su cirujano tomará un historial clínico detallado y examinará el codo para determinar con exactitud la causa de su dolor. Con frecuencia se realizan radiografías para descartar otras patologías; si es necesario, una ecografía o una resonancia magnética pueden mostrar con mayor detalle el estado del tendón.

¿Qué está ocurriendo realmente?

El punto doloroso se encuentra donde un grupo de tendones del antebrazo se fija a esa protuberancia ósea en la parte interna del codo. Imagine esos tendones como una cuerda gruesa compuesta por muchas fibras pequeñas, todas ancladas en un mismo punto. Cada vez que usted agarra algo, realiza un movimiento de balanceo o lanza un objeto, esa “cuerda” tira de su punto de anclaje.

Con el uso repetido y excesivo, se producen pequeñas roturas en dichas fibras. El cuerpo intenta repararlas, pero el daño se genera más rápido de lo que el proceso de curación permite. Con el tiempo, el tejido tendinoso se desgasta, se vuelve más grueso y débil, en lugar de presentar una rotura nítida. Por eso el codo de golfista no es realmente una inflamación ni un hematoma; se trata de un desgaste del propio tendón, lo cual explica por qué el dolor reaparece al volver a realizar las mismas actividades.

Estos tendones no solo sirven para flexionar la muñeca; también refuerzan la parte interna del codo contra la fuerza que se genera al lanzar o al realizar un movimiento brusco. Cuando están desgastados y fatigados, esa función de refuerzo disminuye, y con cada esfuerzo mayor carga se transmite a la zona interna del codo. Esto contribuye al dolor que se siente durante y después de la actividad física.

Esa misma tensión repetida también puede irritar el pequeño nervio que discurre cerca de la parte interna del codo; por eso algunas personas experimentan hormigueo además de dolor en esa zona.

Qué podemos hacer al respecto

El Dr. Kieran Hirpara, cirujano de extremidades superiores en el Mater Private Hospital Rockhampton, comienza con las opciones menos invasivas que se adapten a su condición. Por lo general, los pacientes son derivados a nuestra clínica por su médico de cabecera; si un fisioterapeuta le ha sugerido que nos consulte, igualmente necesitará una derivación de su médico de cabecera para poder acceder al reembolso de Medicare. En su consulta, tomamos su historia clínica, examinamos su codo y solicitamos estudios de imagen si es necesario para confirmar la causa del problema.

En el caso del codo de golfista, normalmente empezamos con reposo y un cambio en las actividades que sobrecarguen el tendón. Esto implica reducir temporalmente el golf, los lanzamientos o el agarre de objetos pesados, para luego volver a incrementar gradualmente dichas actividades. La fisioterapia tiene como objetivo fortalecer los músculos del antebrazo que se insertan en la zona dolorida, de modo que el tendón soporte mejor la carga. La mayoría de las personas mejoran con algún tipo de tratamiento no quirúrgico; vale la pena intentarlo durante 6 meses o más antes de considerar la cirugía. Por lo general, la cirugía se reserva para aquellos síntomas que persisten tras 6 meses o más de tratamiento.

Los analgésicos y los antiinflamatorios no esteroideos (AINE) pueden ayudar a controlar los brotes de dolor mientras se modifican las actividades. No ofrecemos inyecciones para esta afección, por lo que no las trataremos aquí.

Si el tratamiento no quirúrgico no ha producido mejoría suficiente tras 6 meses o más, podríamos hablar sobre la cirugía. El objetivo es liberar o extirpar la parte desgastada y dañada del tendón en su punto de inserción ósea, de modo que el tejido sano pueda asumir su función. Antes de que usted tome cualquier decisión, le explicaremos en detalle en qué consiste la operación y cómo será su recuperación.

Qué esperar

En la mayoría de las personas, el codo de golfista mejora con el tiempo y con el tratamiento adecuado. Vale la pena intentar un tratamiento no quirúrgico durante 6 meses o más. Muchas personas mejoran sin necesidad de operación; sin embargo, el dolor puede tardar en desaparecer y puede reaparecer si se vuelve a realizar el mismo tipo de agarre o movimiento brusco demasiado pronto. Algunas personas siguen sintiendo dolor en el codo un año o más después de su aparición, incluso cuando el tendón ya ha sanado.

Si no se hace ningún cambio, el pronóstico es menos predecible: el dolor tiende a persistir o reaparecer cada vez que se somete el tendón a carga. Cuanto más dure el problema, mayor será el impacto en la capacidad de agarre, en la práctica deportiva y en el trabajo. Por eso recomendamos actuar a tiempo en lugar de esperar a que mejore por sí solo.

Cuando es necesaria la cirugía, el objetivo es lograr un alivio duradero, no un remedio rápido. La liberación del tendón dañado en su punto de anclaje al hueso ha demostrado mejorar significativamente el dolor y la función, con resultados mantenidos durante todo un año. En algunas personas con un problema persistente en el que el tendón se ha desprendido del hueso, la cirugía para extraer el fragmento y reparar el ligamento restaura rápidamente la estabilidad del codo, con pocas complicaciones y buenas valoraciones por parte de los pacientes respecto al resultado.

La recuperación es gradual. En las primeras semanas se busca controlar el dolor y proteger el tendón. En los meses siguientes, los ejercicios de fortalecimiento ayudan a recuperar la capacidad del tendón para soportar el agarre, el levantamiento de pesos y los movimientos bruscos. Algunas personas vuelven a su nivel previo de actividad deportiva o laboral; otras deben modificar ciertas tareas para mantener el codo cómodo.

Es importante tener expectativas realistas: se necesitan meses, no semanas, para la recuperación. La mayoría de las personas logran buenos resultados si reducen las actividades que agravan el problema, completan el programa de fortalecimiento y progresan de forma gradual. Forzar el regreso a la actividad demasiado pronto es la causa más frecuente de recaídas.

¿Cuándo consultar a un especialista?

Acuda a su médico de cabecera si el dolor en la parte interna del codo persiste durante más de unas pocas semanas, si reaparece a pesar del reposo, o si le impide trabajar, jugar al golf o lanzar. Solicite una evaluación por parte de un especialista si siente que su fuerza de agarre es menor que en el otro brazo, si el dolor empeora con el tiempo, o si nota hormigueo a lo largo de la cara interna del codo, ya que el nervio situado allí puede estar irritado junto con el tendón. Los atletas jóvenes que lanzan durante todo el año o en más de un equipo deben someterse a una evaluación temprana; asimismo, cualquier adolescente con dolor en la parte interna del codo durante o después de lanzar necesita ser examinado antes de volver a practicar deporte. Acuda a urgencias si el codo sufrió un golpe directo o una caída, si no puede enderezar el brazo, o si el codo parece estar fuera de su posición normal, pues esto podría indicar una fractura o un desgarro tendinoso que requiere atención inmediata.

En profundidad

Esta sección va más allá de lo necesario para que usted tome sus propias decisiones terapéuticas. Vale la pena leer más sobre el codo de golfista, ya que habitualmente se lo presenta como la versión interna del codo de tenista; sin embargo, en dos aspectos esa descripción resulta engañosa: los factores que lo predisponen son solo parcialmente mecánicos, y la presencia de otro problema simultáneo modifica los resultados que se pueden obtener con la cirugía.

La mayoría de los casos se resuelven sin cirugía

La cifra principal resulta tranquilizadora: en una revisión sobre el dolor medial del codo, el tratamiento conservador mejora el estado de nueve de cada diez pacientes, mientras que la desbridación quirúrgica presenta una tasa de éxito de 80% a 85% [1].

Es importante considerar ambas cifras conjuntamente, no por separado. El tratamiento no quirúrgico tiene una tasa de éxito superior. La cirugía no constituye una versión “mejorada” del mismo tratamiento; es la opción indicada para esa minoría de pacientes en quienes el manejo del tiempo y de la carga física ya no ha surtido efecto, y su tasa de éxito es ligeramente inferior a la del tratamiento de primera línea.

En cuanto al componente de ejercicio, la evidencia respalda que el entrenamiento de fuerza reduce los síntomas en casos de tendinosis; asimismo, las técnicas de manipulación ofrecen un efecto analgésico a corto plazo que podría permitir realizar estiramientos y ejercicios de fortalecimiento más intensos. No obstante, los autores señalan que los resultados aún no son concluyentes [2].

Los factores de riesgo no se limitan únicamente al grado de uso del brazo

La epicondilitis es frecuente en personas en edad laboral; los factores relacionados con la carga física, el tabaquismo y la obesidad son determinantes clave [3]. El tabaquismo y el peso corporal no son aspectos que la mayoría de la gente asocia con problemas tendinosos; ambos influyen en el aporte sanguíneo al tendón y en su entorno metabólico, más que en la magnitud de la tensión que soporta.

Los datos ocupacionales aportan más información. En un estudio realizado con 1,824 trabajadores, se observaron relaciones estadísticamente significativas entre numerosos factores psicosociales personales y laborales, y tanto la epicondilitis medial como la lateral; dichas relaciones se mantuvieron tras ajustar por variables demográficas y exposiciones físicas laborales. Las asociaciones más fuertes fueron entre el agotamiento físico posterior al trabajo y la epicondilitis lateral, con un cociente de probabilidad de 7.04, y entre el agotamiento mental posterior al trabajo y la epicondilitis medial [4].

Ese ajuste es el elemento crucial de la conclusión. No se trata simplemente de que las personas cansadas realicen trabajos más pesados; la relación persiste incluso tras controlar la exposición física. Esto no demuestra que el agotamiento cause tendinopatía, pero sí indica que un plan de tratamiento centrado únicamente en la carga física, sin considerar el grado de agotamiento del paciente tras su jornada laboral, aborda solo una parte del problema.

Por qué el nervio ulnar es clave para el resultado

El dolor medial del codo requiere un amplio diagnóstico diferencial: los trastornos del nervio ulnar, la radiculopatía cervical y las lesiones ligamentosas provocan dolor en el mismo lugar [1]. Esta afección se origina por cargas excéntricas repetitivas y sobrecarga en valgo; inicialmente se trata mediante modificación de actividades y rehabilitación, reservándose la cirugía para los casos con síntomas persistentes [5]. El nervio ulnar es el que más influye en el resultado, ya que discurre justo detrás del origen del tendón que se está tratando.

En los casos en que se realiza desbridamiento, su tasa de éxito puede verse afectada negativamente por una neuritis ulnar concomitante [1]. La consecuencia práctica es que el dolor que persiste tras un desbridamiento realizado técnicamente con éxito no implica necesariamente un fracaso quirúrgico; es posible que el nervio, y no el tendón, haya sido responsable de parte de los síntomas desde el principio. Por ello, la presencia de entumecimiento u hormigueo en el dedo anular y meñique, junto con el dolor en la cara interna del codo, debe comunicarse antes de planificar cualquier intervención, no después.

Referencias

[1] Barco R, Antuña SA. Dolor medial del codo. EFORT Open Rev. 2017;2(8):362-71. https://doi.org/10.1302/2058-5241.2.160006

[2] Hoogvliet P, Randsdorp MS, Dingemanse R, Koes BW, Huisstede BMA. ¿La eficacia de la terapia de ejercicios y las técnicas de movilización sirve como guía para el tratamiento de la epicondilitis lateral y medial? Una revisión sistemática. Br J Sports Med. 2013;47(17):1112-9. https://doi.org/10.1136/bjsports-2012-091990

[3] Shiri R, Viikari-Juntura E, Varonen H, Heliovaara M. Prevalencia y determinantes de la epicondilitis lateral y medial: un estudio poblacional. Am J Epidemiol. 2006;164(11):1065-74. https://doi.org/10.1093/aje/kwj325

[4] Thiese MS, Hegmann KT, Kapellusch J, Merryweather A, Bao S, Silverstein B, et al. Factores psicosociales relacionados con la epicondilitis lateral y medial. J Occup Environ Med. 2016;58(6):588-93. https://doi.org/10.1097/JOM.0000000000000701

[5] Amin NH, Kumar NS, Schickendantz MS. Epicondilitis medial: evaluación y manejo. J Am Acad Orthop Surg. 2015;23(6):348-55. https://doi.org/10.5435/JAAOS-D-14-00145


Evidence & references

This is the clinical evidence summary written for health professionals. It is technical, and it lists the research this page was built from. You do not need to read it to understand your treatment or to make a decision about it.

Overview

  • Percutaneous common flexor origin release of the medial humeral epicondyle is a safe and effective treatment option for golfer's elbow [3].
  • Percutaneous common flexor origin release provides significant and sustainable improvements in pain and function during a 1-year follow-up period [3].
  • Arthroscopic debridement and focused rehabilitation for posterolateral elbow impingement from lateral synovial plicae is highly successful in throwing athletes and golfers [4].
  • Arthroscopic treatment of posterolateral elbow impingement allows athletes to return to their previous level of play [4].
  • Medial epicondylectomy has confirmed success rates between 72% and 94% across 12 studies [41].

Anatomy & Pathophysiology

Bony Anatomy

  • The elbow is a trocho-ginglymoid joint consisting of medial and lateral articulations that provide bony stability [51].
  • The ulnohumeral joint is formed by the articulation of the trochlea with the ulna within the greater sigmoid notch [51].
  • The ulnohumeral joint provides highly congruent anatomy through almost 180° of articular contact, with the exception of a bare area on the greater sigmoid notch devoid of cartilage [51].
  • The coronoid process has medial and lateral facets that buttress the trochlea anteriorly [51].
  • The sublime tubercle is located just distal and medial to the coronoid process and serves as the attachment site for the anterior bundle of the medial ulnar collateral ligament [51].
  • The medial epicondyle is larger and more posteriorly oriented than the lateral epicondyle and forms the attachment site for the origins of the flexor pronator mass [51].
  • The radiocapitellar joint is formed by the articulation of the capitellum and radial head [51].
  • The proximal radioulnar joint holds the radius in close approximation to the ulna via the annular ligament [51].
  • The radial head is a concave elliptical structure covered with articular cartilage along the radiocapitellar joint and approximately 270° of the articular margin [51].
  • The distal humeral articulation is angled 30° from the longitudinal axis of the humerus [51].
  • The axis of rotation is angulated 5° to 7° in the coronal plane relative to the epicondylar axis, with the medial side more distal than the lateral side [51].
  • The ulna medially bends approximately 8° at 8 cm from the tip of the olecranon [51].
  • The articulation to the tip of the coronoid is approximately 30° from the long axis of the ulna in the sagittal plane [51].
  • The articular surface of the distal humerus is angled 30 degrees anterior to the humeral shaft axis [24].
  • The normal range of elbow flexion/extension is 0 to 150 degrees [24].
  • The normal range of forearm pronosupination is 80 to 85 degrees in each direction [24].
  • The functional range of motion for the elbow is 30 to 130 degrees for flexion/extension and 50 degrees for pronosupination [24].
  • The normal valgus carrying angle of the elbow is 5 to 10 degrees for men and 10 to 15 degrees for women [24].
  • In full extension, 60% of axial load is transmitted through the radiocapitellar joint [24].
  • The trochlea has a 300-degree arc of cartilage [54].
  • The medial column of the distal humerus diverges from the humeral shaft at a 45-degree angle [54].
  • The lateral column of the distal humerus diverges from the humeral shaft at a 20-degree angle [54].

Ligamentous Anatomy

  • Elbow stability is determined by primary stabilizers (ulnohumeral articulation, medial ulnar collateral ligament, lateral ulnar collateral ligament) and secondary stabilizers (radiocapitellar articulation, common flexor tendon, common extensor tendon, joint capsule) [17].
  • The medial collateral ligament complex comprises the anterior oblique, posterior oblique, and transverse ligaments [71].
  • The anterior oblique ligament is the strongest component of the medial collateral ligament complex and is the primary stabilizer to valgus stress [71].
  • The anterior oblique ligament originates on the anterior-inferior edge of the medial epicondyle and inserts on the sublime tubercle of the ulna [71].
  • The anterior oblique ligament is composed of anterior and posterior bands that provide reciprocal function in resisting valgus stress [71].
  • The anterior band of the medial collateral ligament is taut in extension, while the posterior band is tight in flexion [71].
  • The anterior bundle of the medial collateral ligament is the primary restraint to valgus stress within functional elbow range of motion [24].
  • The posterior bundle of the medial collateral ligament is the primary restraint to valgus stress with the elbow in maximal flexion [24].
  • Stability in full extension is provided by the medial collateral ligament, joint capsule, and ulnohumeral articulation [24].
  • The medial collateral ligament originates on the posterior medial epicondyle and inserts on the sublime tubercle of the medial coronoid process [24].
  • The lateral ulnar collateral ligament complex originates at the geometric center of the radiocapitellar articulation, just distal to the lateral epicondyle [51].
  • The ulnohumeral articulation contributes to elbow stability, and olecranon resection increases valgus angulation and medial collateral ligament strain during valgus stress [10].

Pathophysiology

  • Medial epicondylar tendinopathy is a pathology of the flexor-pronator muscle group at its origin overlying the medial epicondyle [31].
  • The etiology of medial epicondylar tendinopathy is associated with overuse of the flexor-pronator muscle group [31].
  • Histological analysis of medial epicondylar tendinopathy reveals a brief inflammatory period followed by microtearing, collagen architectural disruption, an incomplete vascular response, and angiofibroblastic degeneration [31].
  • Elbow tendinopathy is a tendon degeneration resulting from continued microtrauma and failed attempts at healing rather than an inflammatory condition [28].
  • Valgus torque generated at the elbow during throwing maneuvers is highest in the late cocking and early acceleration phases of throwing [71].
  • During throwing, the olecranon is repeatedly and forcefully driven into the olecranon fossa, exerting shear forces on the medial aspect of the olecranon tip and the olecranon fossa [10].
  • This process may cause cartilage injury and the development of osteophytes [10].
  • Medial ligamentous laxity commonly exacerbates valgus extension overload syndrome [10].
  • The pathoanatomy of valgus extension overload syndrome includes chondrosis, osteophyte development on the posteromedial olecranon and humerus, and loose bodies [10].
  • The flexor-pronator mass dynamically stabilizes the elbow against valgus torque [62].
  • The medial elbow joint space is significantly reduced under 60-N valgus stress plus 50% maximum voluntary contraction compared to 60-N valgus stress alone [63].
  • Incorporating the pronator teres into contraction tasks significantly reduces the medial joint space, emphasizing the role of the pronator teres in elbow joint stability [64].
  • Repetitive baseball pitching reduces elbow valgus stability, attributed to decreased flexor-pronator mass contractile function [97].
  • Fragmentation of the medial epicondyle may contribute to compromised medial elbow dynamic stability in adult baseball players [93].
  • High elbow varus torque increases the risk of medial elbow disorder [82].
  • A reduction in proximal Hounsfield Unit values of the ulnar collateral ligament may reflect localized structural attenuation that is functionally relevant to medial elbow stability [44].
  • The valgus-hyperextension overloading of the elbow during throwing causes repetitive microtrauma and shear stresses to the medial elbow at the medial epicondyle physis, ulnar collateral ligament, and flexor pronator origin [67].
  • The surrounding elbow musculature, specifically the flexor digitorum superficialis and flexor carpi ulnaris, provide a dynamic stabilizing force across the elbow joint and may be protective of the static restraint of the medial collateral ligament [71].

Classification

  • The Copenhagen Classification of Distal Humeral Fractures (CCDHF) is a classification system designed to distinguish fractures that may not be suitable for open reduction and internal fixation (ORIF) and require treatment with elbow hemiarthroplasty (EHA) or total elbow arthroplasty (TEA) [114].
  • The primary objective of the Copenhagen Classification of Distal Humeral Fractures is to identify patients who may require treatment at a specialized tertiary center where EHA and TEA are available [114].
  • The Wrightington classification system is a tool for characterizing the majority of elbow-fracture dislocations and guiding surgical interventions [56].
  • Accurate diagnosis of medial epicondylitis requires distinguishing it from other elbow conditions, and treatment is guided by the specific pathologic stage of the tendon [9].
  • In a systematic review of medial epicondylitis, elbows were classified as either type Ia/Ib (n = 287; 64.5%) or type IIa/IIb (n = 158; 35.5%) using Gabel-Morrey scoring [21].
  • Concomitant ulnar neuritis was described in 169 elbows (38.0%) in a systematic review of medial epicondylitis where data were available on 445 elbows (92.9%) [21].
  • The most common topic in the contemporary group of the top 100 classical and contemporary papers on elbow surgery was lateral epicondylitis and medial epicondylitis and associated therapies [13].
  • Identification of injury patterns in pediatric humeral medial epicondyle fractures is a key first step in understanding the variability in clinical outcomes with different management strategies for medial elbow injuries [27].

Clinical Presentation

History and Symptoms

  • Patients with medial elbow tendinopathy report a gradual onset of elbow pain localized to the medial epicondyle and over the flexor pronator muscle mass [20].
  • Pain is increased with the offending activity such as throwing or playing golf [20].
  • In the overhead throwing athlete, pain occurring during the acceleration phase over the medial elbow may indicate medial epicondylosis [31].
  • Patients typically present with persistent medial-sided elbow pain that is often localized to the medial epicondyle, with radiation into the proximal forearm [40].
  • Elbow pain is exacerbated by activity and is particularly bothersome during the late cocking phase in overhead throwing or during early acceleration for the thrower, tennis player, or golfer [40].
  • Patient history may include an acute traumatic blow to the elbow resulting in an avulsion of the common flexor tendon [40].
  • More commonly, the pain is characterized by an insidious onset, with persistence despite rest [40].
  • The pain associated with medial epicondylosis is typically insidious in nature and is made worse with specific activities or upper extremity motions for throwing and swinging [31].
  • A history of fluoroquinolone use is associated with increased rates of tendinopathy and rupture [31].
  • Medial epicondylitis is commonly found in occupational settings involving repetitive forceful grip, manual handling of loads 44 lbs (20 kg), or exposure to constant vibratory forces at the elbow [40].
  • In the athlete, medial epicondylitis is typically associated with overhead throwing, golf, or tennis [40].
  • In the literature, medial epicondylitis has been associated with other sports, including football, weightlifting, and bowling [40].
  • The hallmark activity for most medial elbow apophysitis in adolescents is youth baseball [74].
  • A history of repetitive throwing, often year-round or on more than one team, as well as overrepresentation of symptoms in the pitching and catching positions is common in medial elbow apophysitis [74].
  • Poor form and lower-body mechanics during transitional growth years may contribute to an increased valgus position during throwing that increases symptoms in medial elbow apophysitis [74].
  • Pain during and after throwing at the medial elbow is seen in medial elbow apophysitis [74].
  • A large majority of patients with medial epicondyle apophyseal avulsion fractures reported medial elbow pain prior to fracture [15].
  • Medial-sided elbow pain encompasses a significant differential diagnosis, including ulnar neuritis, tendinopathy, ligamentous instability, intra-articular pathology, and trauma [40].
  • Accurate diagnosis of medial epicondylitis requires distinguishing it from other elbow conditions [9].
  • Medial elbow pain is uncommon and requires a broad differential diagnosis including ulnar nerve disorders, cervical radiculopathy, and ligament injuries [19].
  • The patient’s history is critical to differentiating medial epicondylosis from other pathologies on the medial side of the elbow such as valgus extension overload, ulnar neuritis, UCL injury, or even cervical radiculopathy [31].

Physical Examination

  • Physical examination typically reveals tenderness over the flexor pronator origin anterior and distal to the medial epicondyle [20].
  • Pain and weakness on resisted pronation of the forearm have been found to be the most sensitive physical examination findings for medial elbow tendinopathy [20].
  • Pain can also be reproduced with resisted wrist flexion in medial elbow tendinopathy [20].
  • Grip strength can be decreased in medial elbow tendinopathy [20].
  • Focused examination of the medial elbow generally yields pain to palpation over the medial epicondyle [31].
  • Pain with resisted forearm pronation has been described as the most sensitive examination finding for medial epicondylosis [31].
  • Physical examination may detect tenderness 5 to 10 mm distal and anterior to the medial epicondyle that is accompanied by soft-tissue swelling [40].
  • Resisted wrist flexion, forearm pronation, or forceful grip may be weakened compared with that of the contralateral side and may exacerbate elbow pain [40].
  • Patients may present with elbow flexion contracture secondary to pain and guarding [40].
  • Most patients present with normal passive and active range of motion at the elbow and wrist [40].
  • The examination of the athlete with medial elbow pain should include a complete evaluation of the integrity of the ulnar collateral ligament and assessment for ulnar neuritis [20].
  • Ulnar neuritis has been reported in up to 60% of patients ultimately requiring surgery for medial epicondylitis [20].
  • Patients should be evaluated for ulnar neuritis in the setting of medial epicondylosis as 60% of patients requiring surgery have concomitant ulnar neuritis [31].
  • Direct tenderness over the epicondyle is seen in medial elbow apophysitis [74].
  • Tenderness to medial flexor muscle palpation is seen in medial elbow apophysitis [74].
  • Pain with valgus testing is usually less than with direct palpation in medial elbow apophysitis [74].
  • The patient may occasionally present with loss of full elbow extension in medial elbow apophysitis [74].
  • The location, quality or type, context, duration, and severity of elbow pain are important to understanding patients’ pathology and focus the physical examination [68].
  • It is extremely helpful to determine the symptom trajectory, that is, if the pain is getting better, worse, or remaining constant over a period of time [68].

Imaging

  • Plain radiographs are typically normal in medial epicondylitis, although calcifications can sometimes be seen adjacent to the medial epicondyle [20].
  • Plain radiographs of the elbow should always be performed as part of the workup for the etiology of medial-sided elbow pain [31].
  • Given the older age of presentation of most cases of medial epicondylosis, plain radiographs are helpful to rule out arthritis as a possible source of pain [31].
  • A proper diagnosis of medial epicondylosis does not necessarily require advanced imaging [31].
  • Ultrasonography and MRI have the added ability to evaluate the surrounding soft tissues as well as demonstrate objective findings consistent with medial epicondylosis [31].
  • Ultrasonography has a sensitivity of 95% and specificity of 92% in the diagnosis of clinical medial epicondylitis [20].
  • The most common positive ultrasonographic findings in patients with medial epicondylitis were focal hypoechoic regions demonstrating tendinopathy, focal anechoic areas indicating partial common flexor tendon tears, cortical irregularities, and tendon thickening [20].
  • Ultrasonography has a sensitivity of 95% and specificity of 92% with focal hypoechoic areas and intratendinous calcifications representing the typical findings during evaluation of medial epicondylosis [31].
  • MRI has been described as the standard of care for radiographic diagnostic purposes and is extremely helpful if trying to rule out or identify concomitant pathology in medial epicondylosis [31].
  • When reviewing MRI scans for medial epicondylosis a positive finding on the T2-weighted sequence will likely demonstrate intermediate to high signal intensity within the proximal flexor-pronator mass [31].
  • Compared with age-matched control patients, the most specific MRI findings for medial epicondylitis are the presence of intermediate to high T2-weighted signal intensity or high T2-weighted signal intensity within the common flexor tendon and the presence of paratendinous soft-tissue edema [20].
  • The epicondyle may enlarge, exhibit distal traction related avulsive changes, or may have increased apophyseal cartilage width in medial elbow apophysitis [74].
  • While an MRI is often not indicated in the absence of an acute event, edema at the medial epicondyle or sublime tubercle, and occasionally periosteal thickening or layering, may be seen in medial elbow apophysitis [74].

Investigations

Clinical Evaluation

  • Patients with medial elbow tendinopathy report a gradual onset of pain localized to the medial epicondyle and over the flexor pronator muscle mass [20].
  • Pain in medial elbow tendinopathy is increased with offending activities such as throwing or playing golf [20].
  • Physical examination for medial elbow tendinopathy typically reveals tenderness over the flexor pronator origin anterior and distal to the medial epicondyle [20].
  • Pain and weakness on resisted pronation of the forearm are the most sensitive physical examination findings for medial elbow tendinopathy [20].
  • Pain in medial elbow tendinopathy can be reproduced with resisted wrist flexion [20].
  • Grip strength can be decreased in patients with medial elbow tendinopathy [20].
  • The examination of an athlete with medial elbow pain should include a complete evaluation of the integrity of the ulnar collateral ligament [20].
  • The examination of an athlete with medial elbow pain should include an assessment for ulnar neuritis [20].
  • Treatment of medial epicondylitis is guided by the specific pathologic stage of the tendon [9].
  • The physical exam for the elbow is directed by history and the location of the patient's pain in the anterior, posterior, medial, or lateral aspect of the elbow [17].
  • Pathologic entities associated with discrete elbow compartments aid the examiner in detecting pathologic conditions [17].
  • Patients with valgus extension overload syndrome report posteromedial elbow pain that occurs during the deceleration phase of throwing as the elbow reaches terminal extension [10].
  • Pain in valgus extension overload syndrome may also occur during the acceleration phase of throwing [10].
  • Loss of terminal elbow extension may occur in valgus extension overload syndrome [10].
  • Crepitus and tenderness over the posteromedial olecranon may be noted in valgus extension overload syndrome [10].
  • Pain in valgus extension overload syndrome is reproduced when the elbow is forced into extension [10].
  • Elbow flexion contracture may be seen in valgus extension overload syndrome [10].
  • The evaluation of elbow joint instability using fluoroscopy during surgery proved to be valuable for understanding pathology and assessing treatment effectiveness in a case of pediatric medial epicondyle fracture [7].
  • Incarceration of the medial epicondyle in the joint often occurs in association with an elbow dislocation and is important to consider to avoid diagnostic mistakes [33].

Imaging

  • Ultrasonography has a sensitivity of 95% and specificity of 92% for the diagnosis of clinical medial epicondylitis [20].
  • The most common positive ultrasonographic findings in patients with medial epicondylitis are focal hypoechoic regions demonstrating tendinopathy, focal anechoic areas indicating partial common flexor tendon tears, cortical irregularities, and tendon thickening [20].
  • Ultrasonography is recommended as an initial imaging method for the diagnosis of clinical medial epicondylitis [120].
  • The most specific MRI findings for medial epicondylitis are the presence of intermediate to high T2-weighted signal intensity or high T2-weighted signal intensity within the common flexor tendon and the presence of paratendinous soft-tissue edema [20].
  • AP, lateral, oblique, and axillary views of the elbow may reveal posteromedial olecranon osteophytes and/or loose bodies in valgus extension overload syndrome [10].
  • CT with two-dimensional reconstruction and three-dimensional surface rendering best visualizes the pathology of valgus extension overload syndrome [10].
  • MRI may be most helpful in evaluating associated injuries including partial or complete tears of the MCL in valgus extension overload syndrome [10].
  • Radiographs of the elbow should be obtained if the patient has acute traumatic injury or chronic pain [57].
  • CT can be helpful in identifying mineralized intra-articular loose bodies or delineating the anatomy of a complex intra-articular fracture [57].
  • Ultrasonographic soft-tissue evaluation in the elbow is most useful in evaluating the distal biceps and the common flexor and extensor tendons [57].
  • Ultrasonography allows dynamic imaging, which may be useful in evaluating for ulnar nerve subluxation or a snapping triceps [57].
  • MRI is the imaging modality best suited for evaluating soft-tissue structures in the elbow including ligaments, tendons, cartilage, and nerves [57].
  • Conventional MRI sequences should be obtained in all three planes using T1-weighted and fluid-sensitive sequences (short tau inversion recovery or T2-weighted sequences with fat suppression) [57].
  • Magnetic resonance arthrography (MRA) is particularly beneficial in the evaluation of osteochondral lesions, loose bodies, and ulnar collateral ligament (UCL) injury in a throwing athlete [57].
  • Coronal MRI studies should be obtained along a line connecting the medial and lateral epicondyles [57].
  • Sagittal MRI studies should be perpendicular to the coronal studies [57].
  • MRI units with a 3-Tesla magnetic field strength can generate high signal-to-noise ratios and are more able to show normal anatomy than a 1.5-Tesla unit [57].
  • Caution is necessary with 3-Tesla imaging because it can show mild signal alterations of tendons, ligaments, and nerves of the elbow that may not be symptomatic [57].
  • Ligaments and tendons appear anechoic (black) on all MRI imaging sequences [57].
  • With tissue remodeling or degeneration, the signal increases on all MRI sequences [57].
  • Tears are diagnosed on MRI by identifying signal in the tissue that brightens to the level of simple fluid, representing focal discontinuity of tendon or ligament fibers [57].
  • Partial tears on MRI are described by identifying whether the involved pathology occurs at the articular side, intrasubstance, or involves superficial fibers [57].
  • Both partial-thickness and full-thickness tears on MRI should identify whether failure occurs proximally, mid-substance, or distally [57].
  • A combined approach with both MR arthrography and US shows higher accuracy than each modality alone for the assessment of medial elbow pain [79].
  • There is substantial variation in imaging practices across the United States when diagnosing a medial epicondyle fracture [111].
  • CT scans are more likely to be used in smaller cities and older children when diagnosing a medial epicondyle fracture [111].
  • MRI is more likely to be used in smaller hospitals and younger children when diagnosing a medial epicondyle fracture [111].
  • A reduction in proximal Hounsfield Unit values on CT may reflect localized structural attenuation that is functionally relevant to medial elbow stability [44].
  • Providers should take information regarding radiographs missing the real injury into consideration when evaluating medial epicondyle elbow pain in skeletally immature patients [36].
  • Radiographs should always be obtained for the evaluation of elbow stiffness [26].
  • AP, lateral, and oblique radiographs are standard for the evaluation of elbow stiffness [26].
  • Serial radiography is used as follow-up when heterotopic ossification is present in elbow stiffness [26].
  • CT is helpful when assessing for malunion architecture and the location and pattern of osteophytes and/or loose bodies in elbow stiffness [26].
  • Three-dimensional CT is used to check for heterotopic ossification in elbow stiffness [26].
  • CT is not necessary when elbow stiffness is entirely soft-tissue related [26].
  • CT is beneficial when any joint incongruity or abnormal bony anatomy is present in elbow stiffness [26].
  • MRI can be used to evaluate ligaments and tendons in elbow stiffness, but it is rarely indicated [26].
  • Electromyography/nerve conduction velocity studies should be performed if any question about neurologic dysfunction exists in the evaluation of elbow stiffness [26].
  • An assessment for ulnar nerve subluxation should be performed in the evaluation of elbow stiffness [26].

Treatment

Non-Operative Management

  • Nonsurgical treatment for elbow tendinopathy is successful in most cases, with surgical intervention reserved for patients with continued symptoms after 6 months or more of treatment [28].
  • Rest and activity modification are paramount in the nonsurgical management of elbow overuse disorders [28].
  • The current literature provides no definitive recommendations regarding the efficacy of nonsurgical interventions for elbow tendinopathy [28].
  • Regardless of the specific nonsurgical treatment type used, most symptoms improve [28].
  • For valgus extension overload syndrome, nonsurgical treatment includes activity modification with a period of rest from throwing, intra-articular corticosteroid injections, NSAIDs, and a course of dedicated flexor-pronator muscle strengthening [10].
  • Pitching instruction should be started to correct flaws in pitching technique that may contribute to valgus extension overload syndrome [10].
  • For nondisplaced or minimally displaced medial epicondyle fractures (<2 mm) in upper extremity athletes, immobilization in a posterior splint, long-arm cast, or sling for 1 to 2 weeks followed by early active range-of-motion exercises is recommended [85].
  • Following initial immobilization for nondisplaced medial epicondyle fractures, a physical therapy program focusing on strengthening of shoulder, elbow, and wrist muscles associated with throwing should begin at 3 to 4 weeks [85].
  • Wrist flexor strengthening should be avoided for 6 to 8 weeks and any motion causing a valgus moment should be avoided during the rehabilitation of nondisplaced medial epicondyle fractures [85].
  • A throwing program can be initiated at 8 to 12 weeks for nondisplaced medial epicondyle fractures based on radiographic and clinical healing, with no throwing permitted until the fracture site is pain-free [85].
  • Nonoperative treatment may be appropriate for minimally displaced medial epicondylar apophyseal avulsion fractures in youth throwers [116].
  • The outcome of non-operative treatment for medial epicondyle fractures is usually satisfactory, as even a fibrous union is compatible with excellent function [112].

Operative Management: Indications and General Principles

  • Surgical intervention for valgus extension overload syndrome is indicated for patients who continue to have symptoms despite nonsurgical treatment [10].
  • MCL insufficiency is a relative contraindication for isolated olecranon débridement in the treatment of valgus extension overload syndrome [10].
  • Careful evaluation of possible concomitant MCL injury is required before treating valgus extension overload, as treating secondary effects of MCL insufficiency without addressing the underlying MCL pathology leads to unsatisfactory results and increased revision surgery rates [10].
  • Surgical management can be successful in athletes who sustain more significant trauma, have elbow laxity or instability, or have significant fracture fragment displacement in the setting of medial epicondyle fractures [23].
  • Operative treatment of recalcitrant medial epicondylitis is effective in restoring patient function and strength [43].
  • Open and arthroscopic techniques are very effective and comparable for treating chronic medial epicondylitis [46].
  • Arthroscopic surgical treatment for medial epicondylitis of the elbow provides good outcomes and is safe and effective [61].
  • Percutaneous common flexor origin release of the medial humeral epicondyle in golfer's elbow appears to be a safe and effective treatment option providing significant and sustainable improvements in pain and function during a 1-year follow-up period [3].
  • A mini-open muscle resection procedure under local anesthesia for lateral and medial epicondylitis unresponsive to long-term conservative treatments was managed successfully in 41 (97.6%) out of 42 elbows [86].
  • Surgical outcomes for arthroscopic posteromedial decompression of valgus extension overload are generally good, with a cited return to sport rate between 68% and 85% [10].
  • Overaggressive olecranon resection during treatment of valgus extension overload may result in valgus instability of the elbow [10].
  • To prevent increased strain on the MCL during valgus extension overload surgery, it is important to remove only the osteophyte and not the normal olecranon [10].
  • With careful diagnosis and exclusion of other elbow problems, treatment with arthroscopic debridement and focused rehabilitation for posterolateral elbow impingement is highly successful and allows athletes to return to their previous level of play [4].
  • The evaluation of elbow joint instability using fluoroscopy during surgery proved to be valuable for understanding pathology and assessing the effectiveness of treatments in pediatric medial epicondyle fractures with ligament injury [7].
  • Operative treatment affords a significantly higher union rate over the non-operative management of medial epicondyle fractures [94].
  • The procedure of fragment excision and ligament repair for valgus instability due to medial epicondyle nonunion is associated with rapid restoration of elbow stability, minimal surgical morbidity, a high rate of patient satisfaction, and an improvement in objective elbow scores [11].

Operative Management: Specific Procedures

  • Surgical procedures for valgus extension overload include diagnostic elbow arthroscopy, removal of osteophytes on the posteromedial aspect of the olecranon, removal of loose bodies, and débridement of chondromalacia [10].
  • The authors present a surgical technique for arthroscopic extra-articular ulnar nerve release in the setting of stiff elbow applicable to posteromedial elbow pathology by 2 medial portals [6].
  • Surgical techniques currently used for MCL reconstruction include the modified Jobe technique, the docking technique, and the hybrid interference screw technique [10].
  • A muscle-splitting approach is preferred for MCL reconstruction to limit morbidity to the flexor-pronator mass [10].
  • Ulnar nerve transposition is reserved for patients with subluxating nerves or motor weakness in the context of MCL injuries [10].
  • Smith et al proposed treatment of chronic medial epicondyle nonunion by open reduction of the fragment with excision of the fibrinous nonunion tissue and screw fixation with a 3.5 mm or 4.5-mm screw [84].
  • The technique of open reduction and screw fixation for chronic medial epicondyle nonunion is technically challenging because the bony fragment is often too small for this fixation method [84].
  • Five patients required a second procedure for implant removal following the technique of open reduction and screw fixation for chronic medial epicondyle nonunion [84].
  • A suture-augmented lateral ulnar collateral ligament and radial collateral ligament reconstruction provides a reproducible, anatomically based construct that restores posterolateral elbow stability and addresses the complex spectrum of lateral-sided injuries observed in PLRI [29].
  • Strut allograft augmentation restores bone stock in revision elbow arthroplasty, but survivorship free of revision with death as competing risk approaches 75% at 10 years [14].
  • Humeral implants of 10 cm-length could be privileged as first intention implant regardless of the indication for total elbow arthroplasty if there is no imperative to use a longer stem [101].
  • Total elbow arthroplasty is best reserved for low demand, elderly patients who will be able to comply with the 5-lb weightlifting restriction imposed postoperatively to protect the implants from bearing wear, hardware loosening, or failure [75].
  • Open or arthroscopic débridement may be effective in the treatment of early arthritis of the elbow [75].
  • Interposition arthroplasty or total elbow arthroplasty is best reserved for more advanced cases of elbow arthritis [75].
  • Elbow arthrodesis is reserved for patients with painful arthritis who are not candidates for total elbow arthroplasty, especially individuals who place high demands on the upper extremities, such as manual laborers [70].
  • For unilateral arthrodesis of the elbow, a position of 90 to 100 degrees of flexion is desirable to provide the most powerful grip strength [70].
  • If bilateral elbow arthrodesis is indicated, one elbow should be placed in 110 to 120 degrees of flexion to permit the patient to reach the mouth, and the other should be placed in 45 to 65 degrees to aid in personal hygiene [70].
  • For successful elbow arthrodesis, adequate bone stock must be present, although resection of the radial head may be necessary to preserve pronation and supination, and internal or external fixation with bone grafting is typically required [70].
  • The result of interposition arthroplasty in untreated chronic dislocation of the elbow is completely satisfactory, achieving the objective of a minimum range of motion of 100 degrees in addition to elbow stability [8].
  • Both elbow hemi arthroplasty and total elbow arthroplasty provided acceptable elbow function for irreparable distal humeral fractures [1].

Postoperative Rehabilitation

  • Postoperatively for chronic medial epicondyle avulsion treated with fragment excision and ligament reconstruction, the patient is immobilized in a posterior 90 splint for 7 to 10 days until their first postoperative visit [84].
  • The wrist is not necessary to be immobilized to encourage early range of motion following chronic medial epicondyle avulsion surgery [84].
  • Active and active-assisted range of motions are initiated with physical therapy at the first postoperative visit after splint removal for chronic medial epicondyle avulsion [84].
  • No further brace or dynamic immobilization device is used after splint removal for chronic medial epicondyle avulsion [84].
  • The patient is expected to regain full range of motion in the first 3 to 4 weeks after surgery for chronic medial epicondyle avulsion [84].
  • Strengthening is initiated at 6 weeks postoperatively for chronic medial epicondyle avulsion [84].
  • In overhead throwers, an interval throwing program is started at 6 months postoperatively and progresses over 6 weeks for chronic medial epicondyle avulsion [84].
  • Most throwers are able to return to full activities in 6 to 8 months following chronic medial epicondyle avulsion surgery [84].
  • The athlete’s arm is placed in a posterior splint with the elbow immobilized at 90° of flexion for the first 7 days postoperatively following UCL reconstruction to allow early healing of the UCL graft and fascial slings involved in the nerve transposition [110].
  • Following UCL reconstruction, the athlete is progressed from the posterior splint to a hinged elbow ROM brace to protect the healing tissues from valgus stresses that can be detrimental [110].
  • The hinged elbow ROM brace is discontinued at the beginning of week 5 following UCL reconstruction [110].
  • The arm is kept in a splint for 1 week in the immediate postoperative period following combined flexor-pronator and UCL injuries [92].
  • After 1 week, the elbow is managed in a hinged brace for approximately 3 additional weeks following combined flexor-pronator and UCL injuries, allowing motion from 45° of extension to 90° of flexion [92].
  • Motion is slowly advanced to full over the next 5 weeks following combined flexor-pronator and UCL injuries [92].
  • Formal physical therapy begins around 6 weeks and the brace is no longer used following combined flexor-pronator and UCL injuries [92].
  • Patients typically started an interval throwing program at postoperative month 4 following combined flexor-pronator and UCL injuries [92].
  • Players were not allowed to start pitching again competitively until at least 9 months after surgery for combined flexor-pronator and UCL injuries [92].
  • The elbow is maintained in a postsurgical dressing with splint for 5 to 7 days following unilateral interposition arthroplasty of the elbow [87].
  • After initial immobilization, the patient is given a hinged brace and permitted load-free, active motion following unilateral interposition arthroplasty of the elbow [87].
  • Resisted activities, including lifting and pushing, are permitted at 10 to 12 weeks following unilateral interposition arthroplasty of the elbow [87].
  • The patient is placed into a well-padded light splint with the elbow at 90 degrees of flexion and the forearm in pronation following operative treatment of elbow dislocations [108].
  • Ideally, the dressing is removed and motion begun 48 hours after surgery for elbow dislocations unless static joint fixation has been required [108].
  • The elbow should not be immobilized for longer than 2 weeks to avoid excessive stiffness following elbow dislocation surgery [108].
  • Active motion is preferred over passive motion following elbow dislocation surgery as this tends to stabilize the elbow [108].
  • If the MCL is intact and the LCL requires protection, the forearm should be rehabilitated with the forearm in pronation with prosupination only performed at 90 degrees or greater of flexion [108].
  • Varus positioning of the arm should be avoided in patients with LCL injuries and repairs following elbow dislocation surgery [108].
  • If the MCL has been injured but not repaired and the LCL is competent, flexion–extension of the elbow should be performed with the forearm maintained in supination [108].
  • If both the MCL and LCL have been injured, active range of motion should be initiated with the forearm in neutral position [108].
  • Extension is allowed only to the extent that allows congruent tracking intraoperatively following elbow dislocation surgery [108].
  • Passive stretching of the elbow is not performed until ligament healing is progressing, typically beginning 6 weeks postoperatively following elbow dislocation surgery [108].
  • Light strengthening may be started 6 weeks postoperatively with a formal strengthening program initiated at 3 months following elbow dislocation surgery [108].
  • Elbow flexion showed satisfactory recovery on the operated side (135 ± 5°) compared to the contralateral side (138 ± 4°) following biceps brachii tendon reattachment using an adjustable cortical button mechanism, with no statistically significant difference (p 0.212) [91].

Complications

Heterotopic Ossification and Stiffness

  • The reported incidence of heterotopic ossification (HO) after surgical treatment of distal humerus fractures varies from 0% to 49% [98].
  • In a retrospective review of 89 consecutive patients with distal humerus fractures, HO was identified in 37 elbows (42%) [98].
  • HO was associated with less extension and less overall flexion-to-extension movement after distal humerus ORIF [98].
  • Risk factors for elbow stiffness and HO include head injury, polytrauma, severe soft tissue injury, delay to surgical intervention, prolonged postoperative immobilization, and open fractures [98].
  • The development of HO was associated with the method of fracture fixation (perpendicular plating > parallel plating) and the use of bone graft or substitute [98].
  • Most patients with HO do not experience significant functional deficits, so resection is not always necessary [98].
  • Surgical excision of symptomatic HO is associated with significantly better gains in range of motion than release of soft tissue only contractures [98].

Ulnar Collateral Ligament Reconstruction

  • Complications were found in 20% of a cohort undergoing medial ulnar collateral ligament reconstruction, with 4% being major complications including ulnar nerve injuries, medial epicondyle fractures, and revision surgery for osteophyte formation [109].
  • Medial elbow pain during the return-to-throwing period after ulnar collateral ligament reconstruction is not uncommon, with up to half of pitchers potentially experiencing pain [42].

Medial Epicondyle Fractures

  • A large majority of patients with medial epicondyle apophyseal avulsion fractures reported medial elbow pain prior to the fracture [15].
  • At 1 year after initial presentation, bone union of medial epicondylar fragmentation was associated with a decreased prevalence of elbow pain [5].
  • In a study of youth overhead athletes treated with open reduction and internal fixation for medial epicondyle fractures, no major surgical complications were reported, although one patient underwent elective hardware removal [118].

Surgical Procedures for Epicondylitis

  • There were no self-reported differences in complication rates between open (4.4%) and arthroscopic (5.5%) procedures for tennis elbow [39].
  • Percutaneous common flexor origin release of the medial humeral epicondyle in golfer's elbow appears to be a safe treatment option [3].

Recovery

Non-Operative

  • Conservative treatment without prohibiting tennis play resulted in an 83% rate of spontaneous bone union for medial epicondylar fragmentation in male junior tennis players [125].
  • Elbow pain persisted in 50% of subjects at re-examination following conservative treatment for medial epicondylar fragmentation in male junior tennis players [125].
  • The prognosis for medial epicondylitis in occupational settings was good with a 3-year recovery rate at 81% [126].
  • Bone union of the medial epicondylar fragmentation was associated with a decreased prevalence of elbow pain at 1 year after initial presentation in young baseball players [5].

Operative

  • Percutaneous common flexor origin release of the medial humeral epicondyle provides significant and sustainable improvements in pain and function during a 1-year follow-up period [3].
  • Open, anatomical reduction is recommended to ensure restoration of elbow stability for biepicondylar fracture dislocation of a child's elbow [2].
  • Fragment excision and ligament repair for valgus instability due to medial epicondyle nonunion is associated with rapid restoration of elbow stability [11].
  • Fragment excision and ligament repair for valgus instability due to medial epicondyle nonunion is associated with minimal surgical morbidity [11].
  • Fragment excision and ligament repair for valgus instability due to medial epicondyle nonunion is associated with a high rate of patient satisfaction [11].
  • Fragment excision and ligament repair for valgus instability due to medial epicondyle nonunion is associated with an improvement in objective elbow scores [11].
  • After open reduction internal fixation of the medial epicondyle in professional pitchers with a history of ulnar collateral ligament reconstruction, 73.3% were able to return to sport [49].
  • After open reduction internal fixation of the medial epicondyle in professional pitchers with a history of ulnar collateral ligament reconstruction, 55% returned to the same level or higher [49].
  • After open reduction internal fixation of the medial epicondyle in professional pitchers with a history of ulnar collateral ligament reconstruction, there was no significant decline in most performance variables when compared with preoperative performance or matched controls [49].
  • A patient with a medial epicondyle fracture and concomitant flexor-pronator mass avulsion was pain free at the 1-year follow-up visit [32].
  • A patient with a medial epicondyle fracture and concomitant flexor-pronator mass avulsion had symmetric range of motion at the 1-year follow-up visit [32].
  • A patient with a medial epicondyle fracture and concomitant flexor-pronator mass avulsion had elbow stability at the 1-year follow-up visit [32].
  • A patient with a medial epicondyle fracture and concomitant flexor-pronator mass avulsion had function symmetric to the contralateral extremity at the 1-year follow-up visit [32].
  • A patient with a greatly delayed complication of medial epicondyle injury had full range of movement at the elbow at 6 weeks [12].
  • A patient with a greatly delayed complication of medial epicondyle injury had no obvious deformity at 6 weeks [12].
  • A patient with a greatly delayed complication of medial epicondyle injury had no weakness in the limb at 6 weeks [12].
  • Delayed neuropathy of the ulnar nerve associated with elbow dislocation and medial epicondyle fracture appears to be associated with complete recovery in children when promptly treated [127].

Key Evidence

  • [L1] Both treatments provided acceptable elbow function. [1] (10.1016/j.jse.2022.01.016)
  • [L5] They recommend open, anatomical reduction to ensure restoration of elbow stability. [2] (10.1016/s0020-1383(96)00138-6)
  • [L4] Percutaneous common flexor origin release of medial humeral epicondyle in golfer's elbow appears to be a safe and effective treatment option and provides significant and sustainable improvements in pain and function during a 1-year follow-up period. [3] (10.1016/j.rboe.2016.06.007)
  • [L4] With careful diagnosis and exclusion of other elbow problems, treatment with arthroscopic debridement and focused rehabilitation is highly successful and allows these athletes to return to their previous level of play. [4] (10.1177/0363546505281917)
  • [L3] At 1 year after initial presentation, bone union of the medial epicondylar fragmentation was associated with a decreased prevalence of elbow pain. [5] (10.1177/0363546512443807)
  • [L4] The authors present a surgical technique applicable to posteromedial elbow pathology by 2 medial portals. [6] (10.1016/j.eats.2024.103062)
  • [Case_report] The evaluation of elbow joint instability using fluoroscopy during surgery proved to be valuable for both understanding the pathology and assessing the effectiveness of treatments. [7] (10.1016/j.jseint.2024.05.014)
  • [L4] The result is completely satisfactory, achieving the objective of a minimum range of motion of 100 in addition to elbow stability. [8] (10.5435/jaaosglobal-d-21-00034)
  • [L5] Accurate diagnosis requires distinguishing it from other elbow conditions, and treatment is guided by the specific pathologic stage of the tendon. [9] (10.1016/j.csm.2004.04.011)
  • [L4] The procedure is associated with rapid restoration of elbow stability, minimal surgical morbidity, a high rate of patient satisfaction, and an improvement in objective elbow scores. [11] (10.1067/mse.2002.126206)
  • [L5] The patient had full range of movement at the elbow with no obvious deformity at 6 weeks and no weakness in the limb. [12] (10.1016/s0020-1383(98)00141-7)
  • [L5] The most common topic in the classical group was elbow anatomy and function, and the most common topic in the contemporary group was lateral epicondylitis and medial epicondylitis and associated therapies. [13] (10.5435/jaaosglobal-d-23-00287)
  • [L4] Despite early success of this technique for most elbows within the first two tears, survivorship free of revision with death as competing risk approaches 75% at 10 years. [14] (10.1016/j.jseint.2025.101581)
  • [L3] A large majority of patients reported medial elbow pain prior to fracture, suggesting this severe presentation of Little League elbow may be preventable. [15] (10.1177/2325967121s00275)
  • [L5] Medial elbow pain is uncommon and requires a broad differential diagnosis including ulnar nerve disorders, cervical radiculopathy, and ligament injuries. [19] (10.1302/2058-5241.2.160006)
  • [L4] [21] (10.1177/03635465221095565)
  • [L4] Surgical management can be successful in athletes who sustain more significant trauma, who have elbow laxity or instability, or who have significant fracture fragment displacement. [23] (10.1177/0363546513480797)
  • [L4] As the treatment rationale for ME injuries is often predicated on restoring elbow biomechanics through anatomical restoration of the UCL, identification of these injury patterns is potentially a key first step in understanding the variability in clinical outcomes with different management strategies for medial elbow injuries. [27] (10.1177/2325967125s00159)
  • [L5] The described method provides a reproducible, anatomically based construct that restores posterolateral elbow stability and addresses the complex spectrum of lateral-sided injuries observed in PLRI. [29] (10.1016/j.eats.2025.103797)
  • [L5] At the 1-year follow-up visit, the patient was pain free and had symmetric range of motion, elbow stability, and function when compared with his contralateral extremity. [32] (10.2106/jbjs.cc.19.00417)
  • [Case_report] Incarceration of the medial epicondyle in the joint often occurs in association with an elbow dislocation and is important to consider to avoid diagnostic mistakes. [33] (10.1016/j.jse.2011.09.030)
  • [L4] Providers should take this information into consideration when evaluating medial epicondyle elbow pain in skeletally immature patients. [36] (10.1177/2325967126s00147)
  • [L5] [40] (10.5435/JAAOS-D-14-00145)
  • [L5] The article outlines indications and a technique for medial epicondylectomy, noting that 12 studies have confirmed success rates between 72% and 94%. [41] (10.1016/j.hcl.2007.06.002)
  • [L3] Medial elbow pain during the return-to-throwing period after UCLR is not uncommon, with up to half of pitchers potentially experiencing pain. [42] (10.1177/2325967118808782)
  • [L4] Operative treatment of recalcitrant medial epicondylitis is effective in restoring patient function and strength. [43] (10.1308/003588413x13629960048479)
  • [L3] A reduction in proximal HU values may reflect localized structural attenuation that is functionally relevant to medial elbow stability. [44] (10.1177/23259671261472961)
  • [L3] Open and arthroscopic techniques were very effective and comparable for treating chronic medial epicondylitis. [46] (10.1016/j.jse.2022.09.018)
  • [L4] After ORIF of the medial epicondyle in professional pitchers with a history of UCLR, 73.3% were able to return to sport (only 55% at the same level or higher) without a significant decline in most performance variables when compared with their preoperative performance or matched controls. [49] (10.1177/2325967119852896)
  • [L4] The Wrightington classification system is a valuable tool for characterizing the majority of elbow-fracture dislocations and guiding surgical interventions. [56] (10.1016/j.jseint.2024.08.035)
  • [L4] Arthroscopic surgical treatment for medial epicondylitis of the elbow provides good outcomes and is safe and effective. [61] (10.1016/j.jse.2017.08.019)
  • [L5] The flexor-pronator mass dynamically stabilizes the elbow against valgus torque. [62] (10.2106/00004623-200410000-00020)
  • [L5] The medial elbow joint space was significantly reduced under 60-N valgus stress plus 50% MVC compared to 60-N valgus stress alone. [63] (10.1016/j.jse.2022.03.027)
  • [L4] Incorporating the pronator teres into contraction tasks significantly reduced the medial joint space, emphasizing the important role of the PT in elbow joint stability. [64] (10.1016/j.jse.2024.12.025)
  • [L2] The combined approach with both MR arthrography and US shows higher accuracy than each modality alone for the assessment of medial elbow pain. [79] (10.1148/radiol.2015151256)
  • [L3] High elbow varus torque would increase the risk of medial elbow disorder. [82] (10.1177/2325967121s00748)
  • [L4] [84] (10.5435/jaaos-d-17-00446)
  • [L4] Overall, 41 (97.6%) out of 42 elbows with medial or lateral epicondylitis, which were unresponsive to long-term conservative treatments, were managed successfully. [86] (10.4055/cios.2009.1.3.123)
  • [L5] [87] (10.1016/j.eats.2023.09.010)
  • [L4] Elbow flexion showed satisfactory recovery on the operated side (135 ± 5°) compared to the contralateral side (138 ± 4°), with no statistically significant difference (p 0.212). [91] (10.1016/j.jseint.2025.101582)
  • [L4] [92] (10.1177/0363546509351558)
  • [L2] Fragmentation of the medial epicondyle may contribute to compromised medial elbow dynamic stability in adult baseball players. [93] (10.1016/j.xrrt.2026.100680)
  • [L4] Operative treatment affords a significantly higher union rate over the non-operative management of medial epicondyle fractures. [94] (10.1007/s11832-009-0192-7)
  • [L5] Repetitive baseball pitching reduced elbow valgus stability, attributed to decreased flexor-pronator mass contractile function. [97] (10.1016/j.jse.2023.03.026)
  • [L4] Humeral implants of10 cm-length could therefore be privileged as first intention implant regardless of the indication, if there is no imperative to use a longer stem. [101] (10.1016/j.jseint.2025.101575)
  • [L3] There is substantial variation in imaging practices across the United States when diagnosing a medial epicondyle fracture, with CT scans more likely in smaller cities and older children, and MRI more likely in smaller hospitals and younger children. [111] (10.1177/2325967119s00071)
  • [L4] The outcome of non-operative treatment is usually satisfactory as even a fibrous union is compatible with excellent function. [112] (10.1016/0020-1383(88)90109-x)
  • [L4] [114] (10.1016/j.jseint.2024.08.004)
  • [L4] Nonoperative treatment may be appropriate for minimally displaced cases. [116] (10.1177/23259671251365974)
  • [L4] [118] (10.1177/2325967120976573)
  • [L2] Therefore, ultrasonography is recommended as an initial imaging method for the diagnosis of clinical medial epicondylitis. [120] (10.1016/j.apmr.2007.09.048)
  • [L2] Although conservative treatment without prohibiting tennis play resulted in an 83% rate of spontaneous bone union, elbow pain persisted in 50% of subjects at re-examination. [125] (10.1016/j.jse.2014.06.044)
  • [L2] The prognosis for medial epicondylitis in this population was good with a 3-year recovery rate at 81%. [126] (10.1097/01.jom.0000085888.37273.d9)
  • [L4] The delayed neuropathy of the ulnar nerve appears to be associated with a complete recovery in children, as long as it is promptly treated. [127] (10.1016/j.jse.2012.11.009)

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Section 6 -- Term and Termination.

a. This Public License applies for the term of the Copyright and Similar Rights licensed here. However, if You fail to comply with this Public License, then Your rights under this Public License terminate automatically.

b. Where Your right to use the Licensed Material has terminated under Section 6(a), it reinstates:

1. automatically as of the date the violation is cured, provided it is cured within 30 days of Your discovery of the violation; or

2. upon express reinstatement by the Licensor.

For the avoidance of doubt, this Section 6(b) does not affect any right the Licensor may have to seek remedies for Your violations of this Public License.

c. For the avoidance of doubt, the Licensor may also offer the Licensed Material under separate terms or conditions or stop distributing the Licensed Material at any time; however, doing so will not terminate this Public License.

d. Sections 1, 5, 6, 7, and 8 survive termination of this Public License.

Section 7 -- Other Terms and Conditions.

a. The Licensor shall not be bound by any additional or different terms or conditions communicated by You unless expressly agreed.

b. Any arrangements, understandings, or agreements regarding the Licensed Material not stated herein are separate from and independent of the terms and conditions of this Public License.

Section 8 -- Interpretation.

a. For the avoidance of doubt, this Public License does not, and shall not be interpreted to, reduce, limit, restrict, or impose conditions on any use of the Licensed Material that could lawfully be made without permission under this Public License.

b. To the extent possible, if any provision of this Public License is deemed unenforceable, it shall be automatically reformed to the minimum extent necessary to make it enforceable. If the provision cannot be reformed, it shall be severed from this Public License without affecting the enforceability of the remaining terms and conditions.

c. No term or condition of this Public License will be waived and no failure to comply consented to unless expressly agreed to by the Licensor.

d. Nothing in this Public License constitutes or may be interpreted as a limitation upon, or waiver of, any privileges and immunities that apply to the Licensor or You, including from the legal processes of any jurisdiction or authority.


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